INT356379

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Context Info
Confidence 0.54
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.84
Pain Relevance 0.07

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (DMD, Utrn) cytoplasm (DMD, Utrn) cytosol (DMD)
plasma membrane (Utrn) cytoskeleton (DMD)
DMD (Homo sapiens)
Utrn (Mus musculus)
Pain Link Frequency Relevance Heat
fibrosis 36 69.84 Quite High
Inflammation 4 68.08 Quite High
imagery 6 5.00 Very Low Very Low Very Low
Calcium channel 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
tolerance 1 5.00 Very Low Very Low Very Low
Angina 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 6 99.04 Very High Very High Very High
Muscular Dystrophy 140 91.28 High High
Congenital Anomalies 6 88.52 High High
Cardiomyopathy 63 78.24 Quite High
Fibrosis 43 69.84 Quite High
INFLAMMATION 4 68.08 Quite High
Myocardial Infarction 6 66.28 Quite High
Duchenne Muscular Dystrophy 20 5.00 Very Low Very Low Very Low
Dilated Cardiomyopathy 18 5.00 Very Low Very Low Very Low
Death 11 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Whereas it appears that dystrophin is able to fully compensate for the loss of utrophin in the single utrophin knockout mouse, the opposite is not true as there is significant dysfunction in the mdx even if it takes longer to become manifest, so utrophin is not able to fully compensate for the loss of dystrophin.
dystrophin Positive_regulation (compensate) of Negative_regulation (loss) of utrophin associated with targeted disruption
1) Confidence 0.54 Published 2010 Journal The Open Cardiovascular Medicine Journal Section Body Doc Link PMC3024556 Disease Relevance 0.84 Pain Relevance 0.07

General Comments

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