INT36327

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Context Info
Confidence 0.57
First Reported 1987
Last Reported 2003
Negated 0
Speculated 0
Reported most in Abstract
Documents 4
Total Number 5
Disease Relevance 1.72
Pain Relevance 0.52

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Acads) lipid metabolic process (Acads)
Acads (Rattus norvegicus)
Pain Link Frequency Relevance Heat
anesthesia 2 94.16 High High
Pain 3 91.44 High High
cerebral cortex 6 75.00 Quite High
Disease Link Frequency Relevance Heat
Toxicity 3 99.44 Very High Very High Very High
Metabolic Disorder 2 99.32 Very High Very High Very High
Disease 6 97.94 Very High Very High Very High
Rhabdomyolysis 3 92.04 High High
Muscle Weakness 3 81.04 Quite High
Hypertrophic Cardiomyopathy 3 79.56 Quite High
Coma 9 79.28 Quite High
Hypoglycemia 9 78.80 Quite High
Muscle Disease 3 73.60 Quite High
Coronary Heart Disease 3 71.76 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Short-chain acyl-CoA dehydrogenase (SCAD) deficiency is an inherited metabolic disorder biochemically characterized by tissue accumulation of predominantly ethylmalonic acid (EMA) and clinically by neurological dysfunction.
Negative_regulation (deficiency) of SCAD associated with metabolic disorder and anesthesia
1) Confidence 0.57 Published 2003 Journal Neurochem. Res. Section Abstract Doc Link 12716029 Disease Relevance 0.10 Pain Relevance 0.17
Short-chain acyl-CoA dehydrogenase (SCAD) deficiency is an inherited metabolic disorder biochemically characterized by tissue accumulation of predominantly ethylmalonic acid (EMA) and clinically by neurological dysfunction.
Negative_regulation (deficiency) of Short-chain acyl-CoA dehydrogenase associated with metabolic disorder and anesthesia
2) Confidence 0.49 Published 2003 Journal Neurochem. Res. Section Abstract Doc Link 12716029 Disease Relevance 0.10 Pain Relevance 0.17
In two of the disorders, SCAD deficiency and the severe form of MAD deficiency, chronic CNS toxicity is a dominant feature.
Negative_regulation (deficiency) of SCAD associated with toxicity
3) Confidence 0.06 Published 1987 Journal Adv Pediatr Section Abstract Doc Link 3318304 Disease Relevance 0.48 Pain Relevance 0.09
In two of the disorders, SCAD deficiency and the severe form of MAD deficiency, chronic CNS toxicity is a dominant feature.
Negative_regulation (deficiency) of SCAD associated with toxicity
4) Confidence 0.06 Published 1987 Journal Adv Pediatr Section Abstract Doc Link 3318304 Disease Relevance 0.48 Pain Relevance 0.09
There are now nine inherited diseases that have been identified in the pathway of mitochondrial fatty acid oxidation, including LCAD, MCAD, SCAD, and HMG-CoA lyase deficiencies, two forms each of CPT and MAD deficiencies and an incompletely characterized disorder of primary carnitine deficiency.
Negative_regulation (deficiencies) of SCAD associated with disease
5) Confidence 0.03 Published 1987 Journal Adv Pediatr Section Abstract Doc Link 3318304 Disease Relevance 0.56 Pain Relevance 0

General Comments

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