INT38595

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Context Info
Confidence 0.73
First Reported 1984
Last Reported 2004
Negated 1
Speculated 0
Reported most in Abstract
Documents 8
Total Number 8
Disease Relevance 2.90
Pain Relevance 6.25

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell proliferation (BCAR1) cytosol (BCAR1) signal transduction (BCAR1)
cell adhesion (BCAR1) plasma membrane (BCAR1) cell division (BCAR1)
Anatomy Link Frequency
celiac artery 1
BCAR1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Catecholamine 48 100.00 Very High Very High Very High
Opioid 6 99.80 Very High Very High Very High
opiate 4 99.52 Very High Very High Very High
Nicotine 14 99.50 Very High Very High Very High
antagonist 2 98.96 Very High Very High Very High
narcan 14 98.08 Very High Very High Very High
Morphine 8 98.04 Very High Very High Very High
Codeine 16 97.74 Very High Very High Very High
dexamethasone 12 96.36 Very High Very High Very High
Enkephalin 10 94.44 High High
Disease Link Frequency Relevance Heat
Pathologic Constriction 44 100.00 Very High Very High Very High
Mitral Valve Prolapse 32 97.68 Very High Very High Very High
Hypertension 6 97.68 Very High Very High Very High
Pheochromocytoma 6 89.12 High High
Atherosclerosis 10 81.80 Quite High
Disease 1 67.72 Quite High
Syndrome 7 52.88 Quite High
Liver Cancer 1 36.48 Quite Low
Peripheral Arterial Disease 1 7.68 Low Low
Aneurism 1 6.52 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
There was no release of Met-enk and CAs in the medium 10 min after administration, although nicotine did cause a significant release of Met-enk and CAs.
Localization (release) of CAs associated with nicotine and catecholamine
1) Confidence 0.73 Published 1984 Journal Life Sci. Section Abstract Doc Link 6492996 Disease Relevance 0.24 Pain Relevance 0.80
Nicotine (10(-7) M to 10(-4) M) stimulated simultaneous secretion of CAs and Met-enk from the cultured human Ph cells.
Localization (secretion) of CAs associated with catecholamine and nicotine
2) Confidence 0.67 Published 1986 Journal Acta Endocrinol. Section Abstract Doc Link 3788413 Disease Relevance 0.19 Pain Relevance 0.71
In 6, Ph cells were cultured, and the modes of secretion of CAs and Met-enk were examined.
Localization (secretion) of CAs associated with catecholamine
3) Confidence 0.67 Published 1986 Journal Acta Endocrinol. Section Abstract Doc Link 3788413 Disease Relevance 0.15 Pain Relevance 0.49
There was no release of Met-enk and CAs in the medium 10 min after administration, although nicotine did cause a significant release of Met-enk and CAs.
Neg (no) Localization (release) of CAs associated with nicotine and catecholamine
4) Confidence 0.64 Published 1984 Journal Life Sci. Section Abstract Doc Link 6492996 Disease Relevance 0.24 Pain Relevance 0.79
The results of this study demonstrate that CAS is a relatively frequent finding among patients undergoing Doppler sonography of the celiac artery and is frequently associated to MVP.
Localization (finding) of CAS in celiac artery associated with mitral valve prolapse and pathologic constriction
5) Confidence 0.52 Published 2004 Journal Cardiovasc Ultrasound Section Body Doc Link PMC544591 Disease Relevance 1.95 Pain Relevance 0
These results suggest that human Phs may be heterogeneous with regard to storage and secretion of CAs and opioid peptides.
Localization (secretion) of CAs associated with catecholamine and opioid
6) Confidence 0.52 Published 1986 Journal Acta Endocrinol. Section Abstract Doc Link 3788413 Disease Relevance 0.13 Pain Relevance 0.68
Physiological and very high concentrations of morphine have no clear effect on CA release, while an intermediate concentration (38 ng/ml) increases the secretion of all three CAs.
Localization (secretion) of CAs associated with catecholamine and morphine
7) Confidence 0.47 Published 1994 Journal Life Sci. Section Abstract Doc Link 8107518 Disease Relevance 0 Pain Relevance 1.56
Low, physiological concentrations of codeine (500 pg/ml) reduce the release of NE and E, while 8-fold higher concentrations stimulate an instant, transitory release of all three CAs.
Localization (release) of CAs associated with catecholamine and codeine
8) Confidence 0.47 Published 1994 Journal Life Sci. Section Abstract Doc Link 8107518 Disease Relevance 0 Pain Relevance 1.23

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