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Context Info
Confidence 0.31
First Reported 1973
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 4
Total Number 5
Disease Relevance 1.75
Pain Relevance 1.17

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
tendon 1
MRXS5 (Homo sapiens)
Pain Link Frequency Relevance Heat
cINOD 11 99.36 Very High Very High Very High
adenocard 1 97.98 Very High Very High Very High
Analgesic 2 95.96 Very High Very High Very High
Osteoarthritis 47 94.60 High High
Inflammation 34 93.44 High High
metalloproteinase 4 89.12 High High
Inflammatory response 5 80.88 Quite High
cytokine 30 67.12 Quite High
Pain 5 59.36 Quite High
Inflammatory mediators 2 34.16 Quite Low
Disease Link Frequency Relevance Heat
Osteoarthritis 48 94.60 High High
INFLAMMATION 43 93.44 High High
Necrosis 4 86.88 High High
Cancer 5 86.52 High High
Infection 5 75.36 Quite High
Metaplasia 1 73.48 Quite High
Congenital Anomalies 5 69.12 Quite High
Apoptosis 3 61.68 Quite High
Pain 5 59.36 Quite High
Fever 6 59.00 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Their biological function is analogous to cycle adenosine 3",5' monophosphate (cAMP) (i.e., acting as messengers) rather than to a steroid function, although PGs interact with both cAMP and steroids.
PGs Binding (interact) of associated with adenocard
1) Confidence 0.31 Published 1973 Journal J Reprod Med Section Abstract Doc Link 4571712 Disease Relevance 0 Pain Relevance 0.05
The awareness was better amongst senior PGs and SRs.Do you check its availability daily in different ORs?
PGs Binding (senior) of
2) Confidence 0.24 Published 2010 Journal Saudi Journal of Anaesthesia Section Body Doc Link PMC2980666 Disease Relevance 0 Pain Relevance 0
The primary pharmacological action of NSAIDs is, of course, to decrease the formation of PGs and thromboxanes by inhibiting COX, a key enzyme in the biochemical pathway that leads to formation of these potent mediators [24].
PGs Binding (formation) of associated with cinod
3) Confidence 0.02 Published 2004 Journal Crit Care Section Body Doc Link PMC1065065 Disease Relevance 0.56 Pain Relevance 0.78
In contrast to classical PGs, which induce their effects through binding to cell surface G protein-coupled receptors, 15d-PGJ2 induces most of its effects through the nuclear receptor PPAR?.
PGs Binding (binding) of
4) Confidence 0.02 Published 2008 Journal Arthritis Res Ther Section Body Doc Link PMC2656251 Disease Relevance 0.99 Pain Relevance 0.34
It is likely that the extracellular presence of PGs in the septa within the tendon indicates long standing lesions.
PGs Spec (likely) Binding (presence) of in tendon
5) Confidence 0.01 Published 2006 Journal BMC Vet Res Section Body Doc Link PMC1459153 Disease Relevance 0.20 Pain Relevance 0

General Comments

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