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Context Info
Confidence 0.45
First Reported 1982
Last Reported 1982
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.73
Pain Relevance 0.28

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (MTOR) Golgi apparatus (MTOR) endoplasmic reticulum (MTOR)
cytoplasm (MTOR) cytosol (MTOR) signal transduction (MTOR)
Anatomy Link Frequency
neurons 2
MTOR (Homo sapiens)
Pain Link Frequency Relevance Heat
Pain 2 75.00 Quite High
Intractable pain 1 67.44 Quite High
Trigeminal neuralgia 1 61.12 Quite High
diabetic neuropathy 1 58.24 Quite High
nociceptor 1 25.00 Low Low
Disease Link Frequency Relevance Heat
Frailty 2 98.60 Very High Very High Very High
Nociception 3 96.08 Very High Very High Very High
Pain 2 75.00 Quite High
Intractable Pain 1 67.44 Quite High
Headache 1 61.12 Quite High
Diabetic Neuropathy 1 58.24 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Transcutaneous iontophoresis of microtubule inhibitors (Vinblastin, Vincristin, Formyl-Leurosin) in rats induces depletion of fluoride-resistant acid phosphatase (FRAP) and transganglionic degenerative atrophy (trggl. deg. atr.) of the central terminals of primary nociceptive neurons, probably via blockade of axoplasmic transport in the peripheral sensory nerves.
Spec (probably) Positive_regulation (induces) of Spec (probably) Negative_regulation (depletion) of FRAP in neurons associated with nociception and frailty
1) Confidence 0.45 Published 1982 Journal Acta Neurol. Scand. Section Abstract Doc Link 6183918 Disease Relevance 0.73 Pain Relevance 0.28

General Comments

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