INT42942

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Context Info
Confidence 0.67
First Reported 1982
Last Reported 1984
Negated 0
Speculated 0
Reported most in Abstract
Documents 3
Total Number 3
Disease Relevance 1.05
Pain Relevance 0.31

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell adhesion (Clstn2) Golgi apparatus (Clstn2) endoplasmic reticulum (Clstn2)
plasma membrane (Clstn2)
Anatomy Link Frequency
liver 2
Clstn2 (Mus musculus)
Pain Link Frequency Relevance Heat
Parenteral administration 2 93.56 High High
cva 1 89.24 High High
Pain 1 70.96 Quite High
Paracetamol 2 50.44 Quite High
Disease Link Frequency Relevance Heat
Ocular Toxicity (including Many Sub-types) 2 99.08 Very High Very High Very High
Injury 4 95.04 Very High Very High Very High
Hemorrhage 1 89.24 High High
Hepatotoxicity 4 82.12 Quite High
Vascular Disease 1 75.00 Quite High
Pain 1 70.96 Quite High
Paralysis 2 60.80 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The present study suggested that the no-effect level of CS2 exposure on the development of retinopathy was around 2 ppm (8-h time weighted average), and the absence of adverse CS2 effects among the workers studied supported the current standard for occupational CS2 exposure of 10 mg m-3 in China.
Gene_expression (exposure) of CS2 associated with ocular toxicity (including many sub-types)
1) Confidence 0.67 Published 1984 Journal Int Arch Occup Environ Health Section Abstract Doc Link 6332782 Disease Relevance 0.42 Pain Relevance 0.12
Thus CS2 and CS2-producing agents in vivo such as dithiocarbamate derivatives and disulfiram may modify toxicological and pharmacological effects of foreign compounds by inhibiting microsomal drug-metabolizing enzyme activity in the liver.
Gene_expression (producing) of CS2-producing in liver
2) Confidence 0.67 Published 1982 Journal Biochem. Pharmacol. Section Abstract Doc Link 6291543 Disease Relevance 0.31 Pain Relevance 0.10
Thus CS2 and CS2-producing agents in vivo such as dithiocarbamate derivatives and disulfiram may modify toxicological and pharmacological effects of foreign compounds by inhibiting microsomal drug-metabolizing enzyme activity in the liver.
Gene_expression (producing) of CS2 in liver
3) Confidence 0.67 Published 1982 Journal Biochem. Pharmacol. Section Abstract Doc Link 6291543 Disease Relevance 0.31 Pain Relevance 0.10

General Comments

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