INT48775

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Context Info
Confidence 0.41
First Reported 1994
Last Reported 2009
Negated 1
Speculated 1
Reported most in Body
Documents 3
Total Number 4
Disease Relevance 0.15
Pain Relevance 1.55

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Gabbr2) plasma membrane (Gabbr2) cytoplasm (Gabbr2)
signal transducer activity (Gabbr2)
Anatomy Link Frequency
dorsal root 1
brain 1
Gabbr2 (Mus musculus)
Pain Link Frequency Relevance Heat
gABA 29 100.00 Very High Very High Very High
antagonist 6 100.00 Very High Very High Very High
Spinal cord 5 99.40 Very High Very High Very High
Potency 2 96.84 Very High Very High Very High
tetrodotoxin 2 94.04 High High
GABAergic 4 91.36 High High
agonist 6 75.04 Quite High
withdrawal 2 75.00 Quite High
Calcium channel 1 75.00 Quite High
Glutamate 9 72.64 Quite High
Disease Link Frequency Relevance Heat
Nociception 1 75.00 Quite High
Substance Withdrawal Syndrome 1 75.00 Quite High
Attention Deficit Hyperactivity Disorder 4 36.16 Quite Low
Sprains And Strains 43 5.00 Very Low Very Low Very Low
Infection 37 5.00 Very Low Very Low Very Low
Malaria 31 5.00 Very Low Very Low Very Low
Targeted Disruption 28 5.00 Very Low Very Low Very Low
Disease 27 5.00 Very Low Very Low Very Low
Ganglion Cysts 26 5.00 Very Low Very Low Very Low
Stress 13 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
An in vitro mouse hemisected spinal cord was used to characterize the gamma-aminobutyric acid receptor type B (GABA(B)) modulation of the ventral root potential (VRP) in response to electrical stimulation of the dorsal root (DR).
Regulation (modulation) of gamma-aminobutyric acid receptor type B in dorsal root associated with gaba and spinal cord
1) Confidence 0.41 Published 2004 Journal Neurosci. Lett. Section Abstract Doc Link 15135896 Disease Relevance 0.15 Pain Relevance 0.41
We next examined the effect of GABABR activation coupled with a [Ca2+]i increase.
Spec (examined) Regulation (effect) of GABABR
2) Confidence 0.14 Published 2009 Journal Mol Syst Biol Section Body Doc Link PMC2710870 Disease Relevance 0 Pain Relevance 0.05
We find an abundant, but selective, distribution of both RDL and GABABR2 immunoreactivity in the brain.
Regulation (immunoreactivity) of GABABR2 in brain
3) Confidence 0.04 Published 2006 Journal Journal of Insect Science Section Body Doc Link PMC2990333 Disease Relevance 0 Pain Relevance 0.32
Actions evoked by QUAM and HOMOQUAM were not affected by the addition of N-Methyl-D-Aspartate (NMDA) receptor blockers Mg2+ and DL-AP5; non-NMDA antagonist CNQX and GABA B receptor antagonist 2-hydroxysaclofen.
Neg (not) Regulation (affected) of GABA B receptor associated with gaba and antagonist
4) Confidence 0.02 Published 1994 Journal Rev. Esp. Fisiol. Section Abstract Doc Link 7527570 Disease Relevance 0 Pain Relevance 0.77

General Comments

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