INT49198

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Context Info
Confidence 0.30
First Reported 1995
Last Reported 1995
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.20
Pain Relevance 0.16

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signal transduction (Pomc) generation of precursor metabolites and energy (Pomc) extracellular space (Pomc)
extracellular region (Pomc) cell-cell signaling (Pomc) cytoplasm (Pomc)
Pomc (Mus musculus)
Il1 (Mus musculus)
Pain Link Frequency Relevance Heat
narcan 2 79.60 Quite High
antagonist 1 78.56 Quite High
Opioid 1 76.24 Quite High
Disease Link Frequency Relevance Heat
Cancer 1 100.00 Very High Very High Very High
Necrosis 1 100.00 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Exogenous interleukin-1 (IL-1), but neither IL-6 nor tumor necrosis factor (TNF), collaborated with beta-endorphin even in the absence of LPS, suggesting that IL-1 is a primary mediator of the effect of LPS.
beta-endorphin Binding (collaborated) of IL-1 associated with necrosis and cancer
1) Confidence 0.30 Published 1995 Journal Blood Section Abstract Doc Link 7543304 Disease Relevance 0.20 Pain Relevance 0.16

General Comments

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