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Context Info
Confidence 0.05
First Reported 1995
Last Reported 1995
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.20
Pain Relevance 0.16

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intracellular (Gtf3a) DNA binding (Gtf3a) cell-cell signaling (Pomc)
cytoplasm (Pomc) extracellular space (Pomc) generation of precursor metabolites and energy (Pomc)
Pomc (Mus musculus)
Gtf3a (Mus musculus)
Pain Link Frequency Relevance Heat
narcan 2 79.60 Quite High
antagonist 1 78.56 Quite High
Opioid 1 76.24 Quite High
Disease Link Frequency Relevance Heat
Necrosis 1 99.98 Very High Very High Very High
Cancer 1 99.80 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Exogenous interleukin-1 (IL-1), but neither IL-6 nor tumor necrosis factor (TNF), collaborated with beta-endorphin even in the absence of LPS, suggesting that IL-1 is a primary mediator of the effect of LPS.
beta-endorphin Binding (collaborated) of factor associated with necrosis and cancer
1) Confidence 0.05 Published 1995 Journal Blood Section Abstract Doc Link 7543304 Disease Relevance 0.20 Pain Relevance 0.16

General Comments

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