INT49631

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Context Info
Confidence 0.59
First Reported 1995
Last Reported 2010
Negated 1
Speculated 2
Reported most in Body
Documents 14
Total Number 16
Disease Relevance 4.15
Pain Relevance 2.15

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transmembrane transport (Slc43a3)
Anatomy Link Frequency
parietal 3
foot 2
Slc43a3 (Mus musculus)
Pain Link Frequency Relevance Heat
monoamine 4 99.72 Very High Very High Very High
isoflurane 26 99.68 Very High Very High Very High
fluoxetine 12 99.28 Very High Very High Very High
antagonist 6 97.20 Very High Very High Very High
Acute pain 16 95.00 High High
anesthesia 7 94.92 High High
cva 3 89.84 High High
Action potential 26 84.60 Quite High
Gabapentin 1 75.84 Quite High
alcohol 8 75.12 Quite High
Disease Link Frequency Relevance Heat
Anxiety Disorder 664 100.00 Very High Very High Very High
Shock 188 99.68 Very High Very High Very High
Heart Rate Under Development 6 98.06 Very High Very High Very High
Pain 33 95.00 High High
Epilepsy 1 93.20 High High
Convulsion 7 91.68 High High
Cv General 3 Under Development 3 89.84 High High
Cognitive Disorder 31 85.84 High High
Pressure And Volume Under Development 8 84.56 Quite High
Camurati-engelmann Disease 22 72.28 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Behavioral and EEG changes were observed for 30 min following PTZ administration.
Gene_expression (changes) of EEG
1) Confidence 0.59 Published 2010 Journal J. Pharmacol. Sci. Section Abstract Doc Link 20168048 Disease Relevance 0.67 Pain Relevance 0.17
During the transition from W to SWS, they stopped firing before the onset of EEG synchronization (deactivation), with a mean (±SEM) latency of 743.17?
Gene_expression (synchronization) of EEG
2) Confidence 0.17 Published 2010 Journal Frontiers in Behavioral Neuroscience Section Body Doc Link PMC2972729 Disease Relevance 0 Pain Relevance 0
During the transition from waking to sleep, they stopped firing before the onset of EEG synchronization, the first sign of EEG sleep, while, during the transition from sleep to waking, they discharged not before, but after, onset of EEG activation, the first sign of EEG waking (Figure 4).
Gene_expression (synchronization) of EEG
3) Confidence 0.17 Published 2010 Journal Frontiers in Behavioral Neuroscience Section Body Doc Link PMC2972729 Disease Relevance 0 Pain Relevance 0.04
These results show that isoflurane and desflurane produce similar EEG suppression in neurosurgical patients.
Gene_expression (produce) of EEG associated with isoflurane
4) Confidence 0.11 Published 1995 Journal Anesth. Analg. Section Abstract Doc Link 7574015 Disease Relevance 0 Pain Relevance 0.36
It is conceivable that CaMKIV mice have different levels of cortical arousal in response to foot shock which might be detected with EEG.
Gene_expression (detected) of EEG in foot associated with shock
5) Confidence 0.08 Published 2010 Journal Mol Brain Section Body Doc Link PMC2888801 Disease Relevance 0.74 Pain Relevance 0.10
This is the first evidence to show that CaMKIV manipulation can enhance, in a parallel fashion, the expression of fear behavior and 4-7.5 Hz EEG power.
Gene_expression (expression) of EEG associated with anxiety disorder
6) Confidence 0.08 Published 2010 Journal Mol Brain Section Body Doc Link PMC2888801 Disease Relevance 0.31 Pain Relevance 0
We investigated the EEG responses for CaMKIV and WT mice to tone and trace intervals, and following a shock to the foot during the trace fear conditioning day (Figure 4A-F).
Spec (investigated) Gene_expression (responses) of EEG in foot associated with anxiety disorder and shock
7) Confidence 0.07 Published 2010 Journal Mol Brain Section Body Doc Link PMC2888801 Disease Relevance 0.55 Pain Relevance 0
Secondly, EEG frequencies recorded in higher ranges (above 7.5 Hz) were relatively similar between WT and CaMKIV over-expressed mice suggesting that the observation were not a result of differences in electrode positions or differing amounts of contact between the dura and electrode.
Gene_expression (frequencies) of EEG
8) Confidence 0.07 Published 2010 Journal Mol Brain Section Body Doc Link PMC2888801 Disease Relevance 0.07 Pain Relevance 0
Frontal-parietal EEG
Gene_expression (Frontal) of EEG in parietal
9) Confidence 0.07 Published 2010 Journal Mol Brain Section Body Doc Link PMC2888801 Disease Relevance 0.12 Pain Relevance 0.09
Thus, it is likely that the enhancements seen in CaMKIV over-expressed mice with frontal-parietal EEG recordings would also localize to the prelimbic cortex.
Gene_expression (expressed) of EEG in parietal
10) Confidence 0.06 Published 2010 Journal Mol Brain Section Body Doc Link PMC2888801 Disease Relevance 0.66 Pain Relevance 0.03
To examine whether CaMKIV over-expression alters sleep, learning and memory, we performed electroencephalogram (EEG) recordings with frontal-parietal electrodes according to the experimental design outlined in Figure 1.
Spec (examine) Gene_expression (expression) of EEG in parietal
11) Confidence 0.06 Published 2010 Journal Mol Brain Section Body Doc Link PMC2888801 Disease Relevance 0.17 Pain Relevance 0
CaMKIV over-expression and EEG responses to tone, trace interval, and shock
Gene_expression (expression) of EEG associated with shock
12) Confidence 0.06 Published 2010 Journal Mol Brain Section Body Doc Link PMC2888801 Disease Relevance 0.49 Pain Relevance 0
Blast exposure caused the characteristic fall in mean arterial pressure, bradycardia, fast and shallow respiration, and significant increases in both frequency and amplitude of the EEG activity as compared to control animals (25 versus 15–18 Hz, and 22 versus 10??
Gene_expression (activity) of EEG associated with heart rate under development
13) Confidence 0.04 Published 2010 Journal Frontiers in Neurology Section Body Doc Link PMC3009449 Disease Relevance 0.27 Pain Relevance 0.04
Propofol did not produce remarkable epileptiform EEG phenomena at any level of anesthesia.
Neg (not) Gene_expression (produce) of EEG
14) Confidence 0.03 Published 2003 Journal Neurology Section Body Doc Link 14581667 Disease Relevance 0 Pain Relevance 0
Fluoxetine alone was ineffective in reversing EEG slowing produced by reserpine-scopolamine administration.
Gene_expression (produced) of EEG associated with fluoxetine
15) Confidence 0.03 Published 2002 Journal Neuropharmacology Section Abstract Doc Link 11804611 Disease Relevance 0 Pain Relevance 0.66
These data indicate that 5-HT uptake inhibition and concurrent 5-HT(1A) receptor blockade produce a limited normalization of the cortical EEG after monoaminergic-cholinergic blockade.
Gene_expression (produce) of EEG associated with monoamine
16) Confidence 0.03 Published 2002 Journal Neuropharmacology Section Abstract Doc Link 11804611 Disease Relevance 0.09 Pain Relevance 0.66

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