INT50399

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Context Info
Confidence 0.45
First Reported 1995
Last Reported 2003
Negated 1
Speculated 0
Reported most in Abstract
Documents 5
Total Number 5
Disease Relevance 1.75
Pain Relevance 0.32

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (CPN1) extracellular space (CPN1) extracellular region (CPN1)
Anatomy Link Frequency
myocardium 2
soleus 1
CPN1 (Homo sapiens)
Pain Link Frequency Relevance Heat
lidocaine 1 91.24 High High
Angina 5 80.60 Quite High
Inflammation 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Infarction 3 100.00 Very High Very High Very High
Myocardial Infarction 30 96.44 Very High Very High Very High
Angina 3 80.60 Quite High
Cv General 3 Under Development 2 53.28 Quite High
Chlamydia Infection 2 50.00 Quite Low
Coronary Artery Disease 1 50.00 Quite Low
Atherosclerosis 2 5.00 Very Low Very Low Very Low
Infection 2 5.00 Very Low Very Low Very Low
Cardiovascular Disease 1 5.00 Very Low Very Low Very Low
INFLAMMATION 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Neopterin levels, however, were independent of Cpn serostatus when combining both stable and unstable angina patients.
Neg (independent) Positive_regulation (independent) of Cpn
1) Confidence 0.45 Published 2003 Journal Am. Heart J. Section Body Doc Link 12851610 Disease Relevance 0.11 Pain Relevance 0
Stimulation of the CPN proximal to the block elicited a short-latency inhibition of the soleus H-reflex, which was probably mediated by reciprocal Ia interneurones.
Positive_regulation (Stimulation) of CPN in soleus
2) Confidence 0.40 Published 1995 Journal Exp Brain Res Section Abstract Doc Link 7621944 Disease Relevance 0 Pain Relevance 0.09
Our results suggest that the increase of carboxypeptidase N activity after infarction could be induced by an increase in endogenous substrate concentrations, in particular creatine kinase-MB released from damaged myocardium.
Positive_regulation (induced) of carboxypeptidase N in myocardium associated with infarction
3) Confidence 0.16 Published 1997 Journal Eur J Clin Chem Clin Biochem Section Abstract Doc Link 9166972 Disease Relevance 0.55 Pain Relevance 0.08
Our results suggest that the increase of carboxypeptidase N activity after infarction could be induced by an increase in endogenous substrate concentrations, in particular creatine kinase-MB released from damaged myocardium.
Positive_regulation (increase) of carboxypeptidase N in myocardium associated with infarction
4) Confidence 0.11 Published 1997 Journal Eur J Clin Chem Clin Biochem Section Abstract Doc Link 9166972 Disease Relevance 0.55 Pain Relevance 0.08
Furthermore, high baseline levels of carboxypeptidase N will reduce the diagnosis efficiency of creatine kinase-MB isoforms in the diagnosis of acute myocardial infarction.
Positive_regulation (levels) of carboxypeptidase N associated with myocardial infarction
5) Confidence 0.11 Published 1997 Journal Eur J Clin Chem Clin Biochem Section Abstract Doc Link 9166972 Disease Relevance 0.55 Pain Relevance 0.07

General Comments

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