INT51377

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Context Info
Confidence 0.80
First Reported 1993
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 10
Total Number 10
Disease Relevance 3.02
Pain Relevance 2.80

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

carbohydrate metabolic process (Hexb) lysosome (Hexb)
Anatomy Link Frequency
mast cell 1
urine 1
Hexb (Rattus norvegicus)
Pain Link Frequency Relevance Heat
aspirin 12 100.00 Very High Very High Very High
Paracetamol 14 98.76 Very High Very High Very High
anesthesia 15 96.44 Very High Very High Very High
Inflammation 2 95.64 Very High Very High Very High
Glutamate 1 93.36 High High
Cannabinoid receptor 1 85.08 High High
cannabis 1 82.80 Quite High
spastic colon 1 77.84 Quite High
Analgesic 1 76.72 Quite High
Cannabinoid 4 75.00 Quite High
Disease Link Frequency Relevance Heat
Leukemia 7 99.88 Very High Very High Very High
Injury 9 99.80 Very High Very High Very High
Poisoning 1 99.16 Very High Very High Very High
Mast Cell Leukemia 1 98.78 Very High Very High Very High
INFLAMMATION 2 95.64 Very High Very High Very High
Nephrotoxicity 9 95.60 Very High Very High Very High
Disorders Of Creatine Metabolism 1 95.56 Very High Very High Very High
Body Weight 1 93.80 High High
Necrosis 1 93.60 High High
Shock 2 84.92 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Isoniazid-treated, enflurane anesthetized rats developed a 31% reduction in maximal urinary concentrating ability and a 3.5-fold increase in excretion of N-acetyl-beta-glucosaminidase.
Localization (excretion) of N-acetyl-beta-glucosaminidase
1) Confidence 0.80 Published 1993 Journal Anesth. Analg. Section Abstract Doc Link 8214671 Disease Relevance 0.10 Pain Relevance 0.32
In addition these animals showed a sharp increase in urinary excretion of N-acetyl-beta-D-glucosaminidase and alkaline phosphatase.
Localization (excretion) of N-acetyl-beta-D-glucosaminidase
2) Confidence 0.78 Published 1993 Journal Arch Int Physiol Biochim Biophys Section Abstract Doc Link 7691212 Disease Relevance 0.19 Pain Relevance 0.13
One day after anesthesia, sevoflurane-anesthetized rats concentrated urine normally in response to subcutaneous administration of 1-deamino-8-D-arginine vasopressin and exhibited no increase in urinary excretion of N-acetyl beta-glucosaminidase.
Localization (excretion) of N-acetyl beta-glucosaminidase in urine associated with anesthesia
3) Confidence 0.74 Published 1993 Journal Anesth. Analg. Section Abstract Doc Link 8214671 Disease Relevance 0.08 Pain Relevance 0.35
As a marker of renal injury, urinary excretion of N-acetyl-beta-D-glucosaminidase (NAG) and alanine aminopeptidase (AAP) were measured after oral administration of 600 mg/kg of each drug.
Localization (excretion) of N-acetyl-beta-D-glucosaminidase associated with injury
4) Confidence 0.68 Published 1993 Journal Res. Commun. Chem. Pathol. Pharmacol. Section Abstract Doc Link 8105522 Disease Relevance 0.50 Pain Relevance 0.33
N-acetyl-beta-D-glucosaminidase (NAG) and alanine aminopeptidase (AAP) excretion after acute administration of acetaminophen, salsalate and aspirin in rats.
Localization (excretion) of N-acetyl-beta-D-glucosaminidase associated with aspirin and paracetamol
5) Confidence 0.63 Published 1993 Journal Res. Commun. Chem. Pathol. Pharmacol. Section Title Doc Link 8105522 Disease Relevance 0.48 Pain Relevance 0.45
Average values for urinary excretion of protein, beta(2)-microglobulin, and N-acetyl-beta-d-glucosaminidase in the LS group were significantly higher than those in the other groups (LSP, HSP, HS; P < 0.05).
Localization (excretion) of N-acetyl-beta-d-glucosaminidase
6) Confidence 0.41 Published 2001 Journal Anesthesiology Section Body Doc Link 11135718 Disease Relevance 0 Pain Relevance 0
Urinary excretion of protein, albumin, beta(2)-microglobulin, glucose, and N-acetyl-beta-d-glucosaminidase was measured for up to 7 days postoperatively.
Localization (excretion) of N-acetyl-beta-d-glucosaminidase
7) Confidence 0.38 Published 2001 Journal Anesthesiology Section Body Doc Link 11135718 Disease Relevance 0 Pain Relevance 0
Urinary N-acetyl-beta-D glucosaminidase (NAG) excretion increased 4 h after intoxication.
Localization (excretion) of N-acetyl-beta-D glucosaminidase associated with poisoning
8) Confidence 0.31 Published 2010 Journal Pediatr. Nephrol. Section Abstract Doc Link 20352459 Disease Relevance 0.60 Pain Relevance 0.77
Flavone, kaempferol, myricetin and morin were investigated for their action on RBL cell secretion of beta-hexosaminidase stimulated by anti-DNP serum and DNP-BSA, as well as on secretory granule development.
Localization (secretion) of beta-hexosaminidase associated with leukemia
9) Confidence 0.29 Published 1999 Journal Int. J. Immunopharmacol. Section Abstract Doc Link 10405873 Disease Relevance 0.71 Pain Relevance 0.08
By using the rat basophilic leukemia mast cell line (RBL-2H3), we demonstrate that CBD (3-10 muM) augments beta-hexosaminidase release, a marker of cell activation, from antigen-stimulated and unstimulated cells via a mechanism, which is not mediated by G(i)/G(o) protein-coupled receptors but rather is associated with a robust rise in intracellular calcium ([Ca(2+)](i)) levels sensitive to clotrimazole and nitrendipine (10-30 muM).
Localization (release) of beta-hexosaminidase in mast cell associated with mast cell leukemia
10) Confidence 0.25 Published 2007 Journal J. Leukoc. Biol. Section Abstract Doc Link 17339608 Disease Relevance 0.28 Pain Relevance 0.36

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