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Context Info
Confidence 0.12
First Reported 1995
Last Reported 1995
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.85
Pain Relevance 0.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Ins1, Igf1) extracellular region (Ins1, Igf1) carbohydrate metabolic process (Ins1)
cytoplasm (Ins1)
Anatomy Link Frequency
neural 2
Ins1 (Rattus norvegicus)
Igf1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
diabetic neuropathy 2 75.00 Quite High
Pain 1 68.16 Quite High
Spinal cord 1 38.32 Quite Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 7 99.52 Very High Very High Very High
Syndrome 1 78.24 Quite High
Neuropathic Pain 3 75.92 Quite High
Diabetic Neuropathy 2 75.00 Quite High
Weight Loss 1 74.96 Quite High
Eating Disorder 1 73.76 Quite High
Non-diabetic Peripheral Neuropathy 1 68.16 Quite High
Hyperglycemia 2 58.00 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The premise that insulin, IGF-I and IGF-II provide redundant neurotrophic support underlies the following new theory for pathogenesis of diabetic neural disturbances: a loss of insulin activity leads to a secondary partial decline in IGF-I activity.
Negative_regulation (loss) of insulin in neural Spec (partial) Negative_regulation (decline) of IGF-I associated with diabetes mellitus
1) Confidence 0.12 Published 1995 Journal Brain Res. Brain Res. Rev. Section Abstract Doc Link 7711767 Disease Relevance 0.85 Pain Relevance 0.11

General Comments

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