INT52796

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Context Info
Confidence 0.44
First Reported 1995
Last Reported 2000
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 3
Disease Relevance 0
Pain Relevance 1.12

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (Guca2a)
Anatomy Link Frequency
immune cells 1
enteric nervous system 1
Guca2a (Rattus norvegicus)
Pain Link Frequency Relevance Heat
antinociception 4 100.00 Very High Very High Very High
Opioid receptor 1 1 100.00 Very High Very High Very High
Morphine 5 94.40 High High
analgesia 1 84.16 Quite High
opiate 2 75.00 Quite High
Somatostatin 2 60.36 Quite High
Calcitonin gene-related peptide 2 59.32 Quite High
substance P 2 58.00 Quite High
tetrodotoxin 2 53.84 Quite High
Antinociceptive 1 46.88 Quite Low

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Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The developmental changes in mu opiate receptor-mediated antinociception and coupling to guanyl nucleotide binding proteins were studied in the rat.
Regulation (changes) of guanyl associated with antinociception and opioid receptor 1
1) Confidence 0.44 Published 1995 Journal J. Pharmacol. Exp. Ther. Section Abstract Doc Link 7791108 Disease Relevance 0 Pain Relevance 0.84
In conclusion, the present data suggest that the enteric nervous system and immune cells may modulate guanylin release from the rat colon.
Regulation (modulate) of guanylin in enteric nervous system
2) Confidence 0.40 Published 2000 Journal Endocrinology Section Abstract Doc Link 10875263 Disease Relevance 0 Pain Relevance 0.14
In conclusion, the present data suggest that the enteric nervous system and immune cells may modulate guanylin release from the rat colon.
Regulation (modulate) of guanylin in immune cells
3) Confidence 0.14 Published 2000 Journal Endocrinology Section Abstract Doc Link 10875263 Disease Relevance 0 Pain Relevance 0.14

General Comments

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