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Context Info
Confidence 0.45
First Reported 1994
Last Reported 1994
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.36
Pain Relevance 0.40

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (AKR1A1) oxidoreductase activity (AKR1A1)
AKR1A1 (Homo sapiens)
Pain Link Frequency Relevance Heat
alcohol 5 100.00 Very High Very High Very High
tolerance 2 86.32 High High
Analgesic 2 67.04 Quite High
Paracetamol 1 65.68 Quite High
cytokine 1 25.00 Low Low
fibrosis 1 25.00 Low Low
Disease Link Frequency Relevance Heat
Hepatotoxicity 2 98.82 Very High Very High Very High
Injury 1 75.00 Quite High
Toxicity 5 72.56 Quite High
Alcoholic Liver Cirrhosis 2 28.32 Quite Low
Fibrosis 1 25.00 Low Low
Cirrhosis 1 25.00 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Mechanisms of the hepatotoxicity of ethanol are reviewed, including effects resulting from alcohol dehydrogenase (ADH) mediated excessive hepatic generation of NADH and acetaldehyde.
Gene_expression (generation) of alcohol dehydrogenase associated with hepatotoxicity and alcohol
1) Confidence 0.45 Published 1994 Journal Ann. Med. Section Abstract Doc Link 7826592 Disease Relevance 0.15 Pain Relevance 0.08
Redox changes produced by the hepatic alcohol dehydrogenase pathway affect lipid, carbohydrate and protein metabolism.
Gene_expression (produced) of hepatic alcohol dehydrogenase associated with alcohol
2) Confidence 0.03 Published 1994 Journal Alcohol Alcohol Suppl Section Abstract Doc Link 8974351 Disease Relevance 0.20 Pain Relevance 0.31

General Comments

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