INT53309

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Context Info
Confidence 0.39
First Reported 1994
Last Reported 1994
Negated 1
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.84

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Th) mitochondrion (Th) nucleus (Th)
response to stress (Th) cytoplasm (Th)
Anatomy Link Frequency
neurons 2
Th (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Enkephalin 4 100.00 Very High Very High Very High
narcan 2 97.60 Very High Very High Very High
antagonist 1 97.42 Very High Very High Very High
opioid receptor 1 97.20 Very High Very High Very High
Opioid 1 90.04 High High
Calcium channel 1 88.80 High High
Endogenous opioid 2 77.76 Quite High

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Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Pretreatment with the opioid receptor antagonist naloxone (10(-6) M) greatly increased the number of TH-immunoreactive neurons observed in response to KCl treatment, but had no effect on basal or Fsk-stimulated TH immunostaining, nor on basal or stimulated ME release.
Neg (no) Regulation (effect) of Localization (release) of Fsk-stimulated TH in neurons associated with antagonist, narcan, enkephalin and opioid receptor
1) Confidence 0.39 Published 1994 Journal Brain Res. Section Abstract Doc Link 7834330 Disease Relevance 0 Pain Relevance 0.84

General Comments

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