INT54573

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Context Info
Confidence 0.33
First Reported 1994
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 6
Total Number 6
Disease Relevance 2.02
Pain Relevance 0.87

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (NFYB) intracellular (NFYB) DNA binding (NFYB)
Anatomy Link Frequency
brain 1
dendritic cell 1
NFYB (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammatory stimuli 2 99.92 Very High Very High Very High
cytokine 20 99.56 Very High Very High Very High
ischemia 14 97.98 Very High Very High Very High
Inflammation 27 94.64 High High
Central nervous system 5 90.84 High High
tolerance 2 83.96 Quite High
Inflammatory response 3 78.72 Quite High
Spinal cord 2 28.08 Quite Low
Chronic pancreatitis 1 25.00 Low Low
Rsd 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 30 99.76 Very High Very High Very High
Adhesions 4 99.08 Very High Very High Very High
Necrosis 1 98.84 Very High Very High Very High
Cancer 1 98.66 Very High Very High Very High
Reperfusion Injury 5 98.36 Very High Very High Very High
Cv Unclassified Under Development 10 97.98 Very High Very High Very High
Disease 2 95.00 High High
Injury 10 93.80 High High
Infection 5 93.36 High High
Stroke 35 86.76 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In Hap3, following the calculation of MAFs, the analysis procedure (detection of relatives, recalculation of Qmean, etc.) was conducted separately for the subset of Hap3 samples also present in Hap2, for all Hap3 samples, and for samples present in Hap3 only.
Positive_regulation (conducted) of Hap3
1) Confidence 0.33 Published 2010 Journal PLoS Genetics Section Body Doc Link PMC2861700 Disease Relevance 0 Pain Relevance 0
A test of the entire Hap3 cohort (thus including most Hap2 samples) also yielded an insignificant result (Z?
Positive_regulation (entire) of Hap3
2) Confidence 0.33 Published 2010 Journal PLoS Genetics Section Body Doc Link PMC2861700 Disease Relevance 0 Pain Relevance 0
In Hap3, following the calculation of MAFs, the analysis procedure (detection of relatives, recalculation of Qmean, etc.) was conducted separately for the subset of Hap3 samples also present in Hap2, for all Hap3 samples, and for samples present in Hap3 only.
Positive_regulation (conducted) of Hap3
3) Confidence 0.33 Published 2010 Journal PLoS Genetics Section Body Doc Link PMC2861700 Disease Relevance 0 Pain Relevance 0
Oxidants enhance interleukin-1, interleukin-8, and tumor necrosis factor production in response to inflammatory stimuli by activating the nuclear transcription factor, NF kappa B.
Positive_regulation (activating) of nuclear transcription factor associated with necrosis, cancer and inflammatory stimuli
4) Confidence 0.25 Published 1994 Journal New Horiz Section Abstract Doc Link 7922442 Disease Relevance 0.75 Pain Relevance 0.34
We quantified the effect of drugs on the dendritic cell characteristics, such as stimulation of autologous and allogeneic pan-T cell proliferation, surface marker phenotype, IL-12 p40 subunit secretion, and activation of nuclear transcription factor (NF)-kappa B.
Positive_regulation (activation) of nuclear transcription factor in dendritic cell
5) Confidence 0.11 Published 2001 Journal Croat. Med. J. Section Body Doc Link 11471196 Disease Relevance 0 Pain Relevance 0
Studies have shown that ischemia/reperfusion injury induces expression of cell adhesion molecules and cytokines in the brain through activation of the nuclear transcription factor, NF-?
Positive_regulation (activation) of nuclear transcription factor in brain associated with reperfusion injury, ischemia, adhesions and cytokine
6) Confidence 0.01 Published 2010 Journal Journal of Neuroinflammation Section Body Doc Link PMC3022819 Disease Relevance 1.28 Pain Relevance 0.53

General Comments

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