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Context Info
Confidence 0.15
First Reported 1992
Last Reported 1992
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.07

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Acot1) signal transduction (Chrna2) mitochondrion (Acot1)
plasma membrane (Chrna2) lipid metabolic process (Acot1) cytoplasm (Acot1)
Anatomy Link Frequency
neural 2
Acot1 (Rattus norvegicus)
Chrna2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
tetrodotoxin 1 71.48 Quite High


Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
A theoretical model based on these and other findings considers that: AChR activation by spontaneously released ACh is the only neural factor required for the maintenance of G1 + G2 AChE; the amount of A12 AChE is determined by the combined effects of ACh and another neurogenic substance; although mechanisms intrinsic to myofibers control normal levels of G4 AChE, enhanced production of this isoform is initiated through increasing the frequency of ACh-AChR interactions.
Positive_regulation (increasing) of ACh Binding (interactions) of AChR in neural
1) Confidence 0.15 Published 1992 Journal Neurochem. Res. Section Abstract Doc Link 1311432 Disease Relevance 0 Pain Relevance 0.07

General Comments

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