INT56118

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Context Info
Confidence 0.35
First Reported 1994
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 27
Total Number 31
Disease Relevance 6.33
Pain Relevance 0.82

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

pigmentation (EDAR) cell differentiation (EDAR) signal transduction (EDAR)
plasma membrane (EDAR)
Anatomy Link Frequency
macrophages 3
diencephalon 3
embryonic brains 2
sinus 1
spleen 1
EDAR (Homo sapiens)
Pain Link Frequency Relevance Heat
Neuropeptide 385 92.72 High High
chemokine 8 88.00 High High
Inflammation 59 71.44 Quite High
anesthesia 6 50.00 Quite Low
Opioid 105 38.88 Quite Low
substance P 33 36.24 Quite Low
Pain 33 10.00 Low Low
tolerance 30 9.60 Low Low
Enkephalin 63 5.00 Very Low Very Low Very Low
analgesia 30 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Reprotox - General 2 432 100.00 Very High Very High Very High
Aging 12 99.70 Very High Very High Very High
Chronic Disease 490 99.24 Very High Very High Very High
Disease 27 97.32 Very High Very High Very High
Diabetes Mellitus 184 93.32 High High
Insulin Resistance 3 80.92 Quite High
Obesity 3 78.04 Quite High
Metabolic Syndrome 9 76.96 Quite High
INFLAMMATION 32 71.44 Quite High
Increased Venous Pressure Under Development 12 70.80 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Themes only mentioned by HRM professionals were: “a culture of trust, openness and communication within the organization” and “support within the organization”.
Gene_expression (professionals) of HRM
1) Confidence 0.35 Published 2010 Journal J Occup Rehabil Section Body Doc Link PMC2887511 Disease Relevance 0.56 Pain Relevance 0
For group sessions, we used concept mapping to collect information from two groups of HRM professionals and managers.
Gene_expression (professionals) of HRM
2) Confidence 0.35 Published 2010 Journal J Occup Rehabil Section Abstract Doc Link PMC2887511 Disease Relevance 0.28 Pain Relevance 0
Both groups perceive it as important that managers and HRM professionals have knowledge of chronic diseases and of the prevalence of these diseases among employees.
Gene_expression (professionals) of HRM associated with disease and chronic disease
3) Confidence 0.35 Published 2010 Journal J Occup Rehabil Section Body Doc Link PMC2887511 Disease Relevance 0.60 Pain Relevance 0
HRM professionals created two distinct themes: a “company culture that allows for trust, openness and communication between workers and management” and “a supportive organization”.
Gene_expression (professionals) of HRM
4) Confidence 0.35 Published 2010 Journal J Occup Rehabil Section Body Doc Link PMC2887511 Disease Relevance 0.47 Pain Relevance 0
This study revealed HRM professionals and managers are similar in some aspects and differ in other aspects with respect to what they perceive as important for enabling sustained employability of chronically ill employees in the work-place.
Gene_expression (professionals) of HRM associated with chronic disease
5) Confidence 0.35 Published 2010 Journal J Occup Rehabil Section Body Doc Link PMC2887511 Disease Relevance 0.53 Pain Relevance 0
Both peptides were more highly expressed in ed17 but only SGKLSFLEDE was significantly affected over all groups (F-test statistics, adjusted p < 0.001) with a significant interaction effect and near significant EE2 treatment effect (p-value 0.062).
Gene_expression (expressed) of ed17
6) Confidence 0.33 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0.08
The diencephalon was excised as quickly as possible from embryonic brains on ed12 and ed17 and was placed in pre-weighed Eppendorf tubes.
Gene_expression (excised) of ed12 in embryonic brains
7) Confidence 0.33 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0.03
The diencephalon was excised as quickly as possible from embryonic brains on ed12 and ed17 and was placed in pre-weighed Eppendorf tubes.
Gene_expression (excised) of ed17 in embryonic brains
8) Confidence 0.33 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0.03
A clear separation between ed12 and ed17 peptide expression was visible in a hierarchical clustering (Figure 1A) of all eight experimental groups with the majority of the identified peptides being more highly expressed in ed17 than in ed12 diencephalon (Figure 1B).
Gene_expression (expression) of ed12 in diencephalon
9) Confidence 0.30 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0.06
A clear separation between ed12 and ed17 peptide expression was visible in a hierarchical clustering (Figure 1A) of all eight experimental groups with the majority of the identified peptides being more highly expressed in ed17 than in ed12 diencephalon (Figure 1B).
Gene_expression (expression) of ed17 in diencephalon
10) Confidence 0.30 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0.06
The samples were ed12 male controls (n = 5), ed12 female controls (n = 4), ed12 male EE2 exposed (n = 4), ed12 female EE2 exposed (n = 4), ed17 male controls (n = 4), ed17 female controls (n = 4), ed17 male EE2 exposed (n = 4) and ed17 female EE2 exposed (n = 5).
Gene_expression (exposed) of ed12
11) Confidence 0.29 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0.11
The data was grouped into eight groups: ed12 male controls (ed12MC, n = 5), ed12 female controls (ed12FC, n = 5), ed12 male EE2 (ed12ME, n = 5), ed12 female EE2 (ed12FE, n = 4), ed17 male controls (ed17MC, n = 4)), ed17 female controls (ed17FC, n = 4), ed17 male EE2 (ed17ME, n = 4), and ed17 female EE2 (ed17FE, n = 5) (Table 4).
Gene_expression (controls) of ed12MC
12) Confidence 0.29 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0
The data was grouped into eight groups: ed12 male controls (ed12MC, n = 5), ed12 female controls (ed12FC, n = 5), ed12 male EE2 (ed12ME, n = 5), ed12 female EE2 (ed12FE, n = 4), ed17 male controls (ed17MC, n = 4)), ed17 female controls (ed17FC, n = 4), ed17 male EE2 (ed17ME, n = 4), and ed17 female EE2 (ed17FE, n = 5) (Table 4).
Gene_expression (controls) of ed17ME
13) Confidence 0.29 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0
The fact that most identified peptides were more highly expressed in ed17 compared to ed12 was not representative for all plotted peptides (Figure 1A-B, Table 2).
Gene_expression (expressed) of ed17
14) Confidence 0.29 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0.07
The data was grouped into eight groups: ed12 male controls (ed12MC, n = 5), ed12 female controls (ed12FC, n = 5), ed12 male EE2 (ed12ME, n = 5), ed12 female EE2 (ed12FE, n = 4), ed17 male controls (ed17MC, n = 4)), ed17 female controls (ed17FC, n = 4), ed17 male EE2 (ed17ME, n = 4), and ed17 female EE2 (ed17FE, n = 5) (Table 4).
Gene_expression (controls) of ed17MC
15) Confidence 0.29 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0
The data was grouped into eight groups: ed12 male controls (ed12MC, n = 5), ed12 female controls (ed12FC, n = 5), ed12 male EE2 (ed12ME, n = 5), ed12 female EE2 (ed12FE, n = 4), ed17 male controls (ed17MC, n = 4)), ed17 female controls (ed17FC, n = 4), ed17 male EE2 (ed17ME, n = 4), and ed17 female EE2 (ed17FE, n = 5) (Table 4).
Gene_expression (controls) of ed12ME
16) Confidence 0.29 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0
The samples were ed12 male controls (n = 5), ed12 female controls (n = 4), ed12 male EE2 exposed (n = 4), ed12 female EE2 exposed (n = 4), ed17 male controls (n = 4), ed17 female controls (n = 4), ed17 male EE2 exposed (n = 4) and ed17 female EE2 exposed (n = 5).
Gene_expression (exposed) of ed12
17) Confidence 0.29 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0.11
The samples were ed12 male controls (n = 5), ed12 female controls (n = 4), ed12 male EE2 exposed (n = 4), ed12 female EE2 exposed (n = 4), ed17 male controls (n = 4), ed17 female controls (n = 4), ed17 male EE2 exposed (n = 4) and ed17 female EE2 exposed (n = 5).
Gene_expression (exposed) of ed17
18) Confidence 0.29 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0.10
The data was grouped into eight groups: ed12 male controls (ed12MC, n = 5), ed12 female controls (ed12FC, n = 5), ed12 male EE2 (ed12ME, n = 5), ed12 female EE2 (ed12FE, n = 4), ed17 male controls (ed17MC, n = 4)), ed17 female controls (ed17FC, n = 4), ed17 male EE2 (ed17ME, n = 4), and ed17 female EE2 (ed17FE, n = 5) (Table 4).
Gene_expression (controls) of ed12
19) Confidence 0.29 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0
Overall, the data indicates that the ed12 quail diencephalon shows a more distinct EE2 sensitive and sex peptide expression pattern as compared to ed17 (Figure 1, Additional file 3).
Gene_expression (expression) of ed17 in diencephalon
20) Confidence 0.29 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2851587 Disease Relevance 0 Pain Relevance 0.06

General Comments

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