INT56984

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Context Info
Confidence 0.06
First Reported 1993
Last Reported 1993
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.07
Pain Relevance 0.25

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signal transduction (Gabbr1) extracellular region (CSN3) plasma membrane (Gabbr1)
transcription factor binding (Gabbr1) cytoplasm (Gabbr1) signal transducer activity (Gabbr1)
Gabbr1 (Mus musculus)
CSN3 (Homo sapiens)
Pain Link Frequency Relevance Heat
Action potential 3 97.64 Very High Very High Very High
antagonist 1 85.68 High High
agonist 1 79.88 Quite High
depression 1 75.00 Quite High
tetrodotoxin 1 45.20 Quite Low
Spinal cord 2 35.48 Quite Low
Eae 1 25.00 Low Low
Disease Link Frequency Relevance Heat
Depression 1 75.00 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Intracellular injection of the GTP analogue GTP gamma S also reduced the duration of the action potential and the peak amplitude of the AHP in TEA, thus supporting the notion that a GTP-binding protein (G-protein)-mediated GABAB receptor activation reduced the calcium inflow, leading to less activation of KCa channels and, consequently, to a smaller peak amplitude of the AHP. 4.
Positive_regulation (activation) of GABAB receptor Positive_regulation (leading) of Positive_regulation (activation) of KCa associated with action potential
1) Confidence 0.06 Published 1993 Journal J. Neurophysiol. Section Abstract Doc Link 8120601 Disease Relevance 0.07 Pain Relevance 0.25

General Comments

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