INT57389

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Context Info
Confidence 0.45
First Reported 1993
Last Reported 1998
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.28
Pain Relevance 0.78

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (ARL4C) plasma membrane (ARL4C) nucleus (ARL4C)
intracellular (ARL4C) GTPase activity (ARL4C) cytoplasm (ARL4C)
Anatomy Link Frequency
LAK cell 2
ARL4C (Homo sapiens)
Pain Link Frequency Relevance Heat
cINOD 8 98.54 Very High Very High Very High
aspirin 3 86.80 High High
cytokine 2 84.36 Quite High
Inflammation 1 72.28 Quite High
Disease Link Frequency Relevance Heat
INFLAMMATION 4 75.00 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These results indicated that PGE2 suppression may not be the only mechanism of indomethacin for upregulation of LAK cell activity, and even immune functions.
Positive_regulation (upregulation) of LAK in LAK cell
1) Confidence 0.45 Published 1993 Journal Proc. Natl. Sci. Counc. Repub. China B Section Abstract Doc Link 8171164 Disease Relevance 0.06 Pain Relevance 0.14
These two NSAIDs augmented proliferation of phytohemagglutinin (PHA)-stimulated PBMC and enhanced NK and LAK cell activities.
Positive_regulation (enhanced) of LAK in LAK cell associated with cinod
2) Confidence 0.45 Published 1998 Journal Immunopharmacology Section Abstract Doc Link 9858064 Disease Relevance 0.22 Pain Relevance 0.63

General Comments

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