INT57469

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Context Info
Confidence 0.24
First Reported 1994
Last Reported 1994
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 3
Disease Relevance 0.51
Pain Relevance 2.02

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nuclear chromosome (ZNF238) nucleus (ZNF238) intracellular (ZNF238)
DNA binding (ZNF238)
Anatomy Link Frequency
SK-N-SH 3
ZNF238 (Homo sapiens)
Pain Link Frequency Relevance Heat
Enkephalin 9 99.82 Very High Very High Very High
narcan 6 99.82 Very High Very High Very High
Morphine 12 99.52 Very High Very High Very High
agonist 6 90.60 High High
Disease Link Frequency Relevance Heat
Neuroblastoma 3 97.80 Very High Very High Very High
Bordatella Infection 3 95.36 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These data suggest that d receptor-stimulation increases tyrosine phosphorylation of the 58 kDa protein through Gi protein in SK-N-SH cells.
Phosphorylation (phosphorylation) of 58 kDa protein in SK-N-SH
1) Confidence 0.24 Published 1994 Journal Biochem. Biophys. Res. Commun. Section Abstract Doc Link 8179614 Disease Relevance 0.17 Pain Relevance 0.65
A 58 kDa protein which was phosphorylated on tyrosine residues with morphine was found in human neuroblastoma cells (SK-N-SH cells) by immunoblot with monoclonal anti-phosphotyrosine antibody.
Phosphorylation (phosphorylated) of 58 kDa protein in SK-N-SH associated with neuroblastoma and morphine
2) Confidence 0.24 Published 1994 Journal Biochem. Biophys. Res. Commun. Section Abstract Doc Link 8179614 Disease Relevance 0.16 Pain Relevance 0.57
Tyrosine phosphorylation of a 58 kDa protein induced by morphine in SK-N-SH cells.
Phosphorylation (phosphorylation) of 58 kDa protein in SK-N-SH associated with enkephalin and morphine
3) Confidence 0.24 Published 1994 Journal Biochem. Biophys. Res. Commun. Section Title Doc Link 8179614 Disease Relevance 0.17 Pain Relevance 0.80

General Comments

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