INT57954

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Context Info
Confidence 0.11
First Reported 1993
Last Reported 2006
Negated 0
Speculated 0
Reported most in Abstract
Documents 4
Total Number 5
Disease Relevance 4.51
Pain Relevance 0.89

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Anxa3) cytoplasm (Anxa3)
Anatomy Link Frequency
plasma 1
VCS 1
heart 1
Anxa3 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
cva 7 95.00 High High
Pain 4 91.60 High High
Angina 4 83.80 Quite High
Disease Link Frequency Relevance Heat
Thrombosis 7 99.68 Very High Very High Very High
Congenital Anomalies 2 99.04 Very High Very High Very High
Obesity 1 98.78 Very High Very High Very High
Heart Disease 1 98.60 Very High Very High Very High
Neurological Disease 1 98.30 Very High Very High Very High
Malignant Neoplastic Disease 1 97.86 Very High Very High Very High
Disease 2 97.20 Very High Very High Very High
Pulmonary Embolism 7 95.00 High High
Syndrome 6 94.88 High High
Infarction 4 94.40 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The patient had none of the following risk factors for pulmonary emboli: malignancy, neurological disease, heart disease, obesity, pregnancy, or a congenital coagulative abnormality such as deficiency of AT-III, protein C, protein S, or plasminogen.
Negative_regulation (deficiency) of AT-III in heart associated with malignant neoplastic disease, neurological disease, heart disease and obesity
1) Confidence 0.11 Published 1996 Journal Nihon Kyobu Shikkan Gakkai Zasshi Section Abstract Doc Link 8622276 Disease Relevance 1.02 Pain Relevance 0.27
The investigation of her family revealed that her son also showed decrease AT-III activity, indicating a congenital abnormality. 26 families with this disease have been reported in Japan.
Negative_regulation (decrease) of AT-III associated with congenital anomalies and disease
2) Confidence 0.10 Published 1993 Journal Kokyu To Junkan Section Abstract Doc Link 8235115 Disease Relevance 0.97 Pain Relevance 0.20
AT-III activity was found to be only 50% of normal value, indicating that the pulmonary thromboemboli were due to AT-III deficiency.
Negative_regulation (deficiency) of AT-III
3) Confidence 0.07 Published 1993 Journal Kokyu To Junkan Section Abstract Doc Link 8235115 Disease Relevance 0.85 Pain Relevance 0.25
Antithrombin III (AT-III), responsible for a large part of thrombin inactivation capacity in plasma, is the most powerful inhibitor of the thrombosis process.
Negative_regulation (inhibitor) of AT-III in plasma associated with thrombosis
4) Confidence 0.03 Published 2006 Journal Clin. Nephrol. Section Abstract Doc Link 16429847 Disease Relevance 0.86 Pain Relevance 0.08
At advanced examinations due to lack of response to heparin and clinical worsening, VCS and AT-III deficiency were determined.
Negative_regulation (deficiency) of AT-III in VCS
5) Confidence 0.03 Published 2006 Journal Clin. Nephrol. Section Abstract Doc Link 16429847 Disease Relevance 0.81 Pain Relevance 0.09

General Comments

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