INT58567

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Context Info
Confidence 0.21
First Reported 1994
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0
Pain Relevance 1.21

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (Prss12)
Anatomy Link Frequency
bile 2
Prss12 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Bile 11 99.84 Very High Very High Very High
antagonist 2 98.52 Very High Very High Very High
tetrodotoxin 2 95.84 Very High Very High Very High
Kinase C inhibitor 1 94.16 High High
Action potential 2 93.76 High High
Calcium channel 3 90.24 High High
GABAergic 5 82.76 Quite High
Hippocampus 3 75.00 Quite High
Pyramidal cell 1 71.88 Quite High

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Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In contrast, the micellar form of oleic acid (20 and 100 mM) in bile extract (1%) or TC (20 mM) dose dependently reduced the release of NT-LI induced by bile extract or TC alone.
Negative_regulation (reduced) of Positive_regulation (induced) of NT in bile associated with bile
1) Confidence 0.21 Published 1994 Journal Endocrinology Section Abstract Doc Link 8299558 Disease Relevance 0 Pain Relevance 0.50
NT-induced increase in sIPSC frequency was blocked by application of the specific NTS1 antagonist SR48692, the phospholipase C (PLC) inhibitor U73122, the IP(3) receptor antagonist 2-APB, and the protein kinase C inhibitor GF109203X, suggesting that NT increases gamma-aminobutyric acid release via a PLC pathway.
Negative_regulation (blocked) of Positive_regulation (increase) of NT-induced associated with antagonist and kinase c inhibitor
2) Confidence 0.19 Published 2008 Journal J. Neurophysiol. Section Abstract Doc Link 18337367 Disease Relevance 0 Pain Relevance 0.71

General Comments

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