INT61698

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Context Info
Confidence 0.05
First Reported 1996
Last Reported 1996
Negated 1
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.88

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Abat) protein modification process (G2e3) Golgi apparatus (G2e3)
intracellular (G2e3) cytoplasm (G2e3)
Anatomy Link Frequency
brain 2
G2e3 (Rattus norvegicus)
Abat (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Morphine 6 100.00 Very High Very High Very High
gABA 4 100.00 Very High Very High Very High
Action potential 1 99.28 Very High Very High Very High
Neurotransmitter 1 94.68 High High
Analgesic 1 93.92 High High

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Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Addition of M3G or morphine in a range of concentrations (0.1-10 microM) to the superfusion medium did not alter the K+-evoked release of [14C]-GABA or [3H]-glutamic acid from rat brain synaptosomes, suggesting that the CNS excitation observed following central administration of M3G (2-5) and supra-analgesic doses of morphine (2,3,6,7) does not occur by a generalized inhibition of GABA release or facilitation of glutamic acid release from pre-synaptic nerve terminals.
M3G Neg (not) Regulation (alter) of Localization (release) of GABA in brain associated with gaba, analgesic and morphine
1) Confidence 0.05 Published 1996 Journal Life Sci. Section Abstract Doc Link 8594310 Disease Relevance 0 Pain Relevance 0.88

General Comments

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