INT63064

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Context Info
Confidence 0.50
First Reported 1996
Last Reported 2010
Negated 2
Speculated 2
Reported most in Body
Documents 7
Total Number 8
Disease Relevance 2.66
Pain Relevance 0.65

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (PRM1) nucleoplasm (PRM1) chromosome (PRM1)
nucleus (PRM1) DNA binding (PRM1)
Anatomy Link Frequency
muscle 1
PRM1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 11 93.08 High High
melanocortin 1 receptor 4 73.68 Quite High
addiction 6 73.28 Quite High
fibrosis 33 73.16 Quite High
Bioavailability 2 69.56 Quite High
cytokine 2 66.56 Quite High
peripheral neuropathy 2 50.00 Quite Low
corticosteroid 2 39.36 Quite Low
cINOD 1 32.52 Quite Low
anesthesia 1 10.00 Low Low
Disease Link Frequency Relevance Heat
Asthma 41 98.28 Very High Very High Very High
Coronary Artery Disease 34 95.84 Very High Very High Very High
INFLAMMATION 12 93.08 High High
Disease 23 87.08 High High
Rupture 4 76.68 Quite High
Apoptosis 3 73.56 Quite High
Cystic Fibrosis 33 73.16 Quite High
Contagious Ecthyma 6 72.76 Quite High
Neuropathy 116 71.72 Quite High
Cytomegalovirus Infection 3 71.28 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These researchers observed that the P1 peak was absent in 6 of 11 patients with life-threatening asthma, whereas it was present in 14 of the 15 control asthmatic patients and in all the 14 nonasthmatic patients.
Neg (absent) Gene_expression (absent) of P1 associated with asthma
1) Confidence 0.50 Published 2007 Journal Respir Res Section Body Doc Link PMC2020473 Disease Relevance 1.65 Pain Relevance 0.24
P has_part P1 = [definition] for all p, if Pp then there is some p1 such that P1p1 and p1 part_of p
Gene_expression (such) of p1
2) Confidence 0.41 Published 2005 Journal Genome Biol Section Body Doc Link PMC1175958 Disease Relevance 0 Pain Relevance 0
flanking sequences of the JUNB gene were amplified by PCR, fused to MC4R sequences downstream of P1 replacing the MC4R 3?
Gene_expression (replacing) of P1
3) Confidence 0.12 Published 2010 Journal EMBO J Section Body Doc Link PMC2876958 Disease Relevance 0.14 Pain Relevance 0.30
In case of existence of such association our further aim was to verify and explain this phenomenon by examination of promoter P1 of IGF-1gene and receptor gene for IGF-1.
Spec (examination) Gene_expression (examination) of P1
4) Confidence 0.07 Published 2010 Journal Protein J Section Abstract Doc Link PMC2992669 Disease Relevance 0.36 Pain Relevance 0
In case of existence of such association our further aim was to verify and explain this phenomenon by examination of promoter P1 of IGF-1gene and receptor gene for IGF-1.
Spec (examination) Gene_expression (examination) of P1
5) Confidence 0.07 Published 2010 Journal Protein J Section Body Doc Link PMC2992669 Disease Relevance 0.38 Pain Relevance 0.10
During DF/SLR the mean intensity went from 0.8 ± 1.5 at the START position to 3.0 ± 1.8 at P1 and to 6.8 ± 2.0 at P2 (p < 0.001 both).
Gene_expression (position) of P1
6) Confidence 0.05 Published 2010 Journal BMC Neurol Section Body Doc Link PMC2940775 Disease Relevance 0 Pain Relevance 0
Using the conservative threshold of 1.5-fold increase over resting levels, no muscle activity was triggered at the START position, P1 or P2 during PF/SLR.
Gene_expression (position) of P1 in muscle
7) Confidence 0.05 Published 2010 Journal BMC Neurol Section Body Doc Link PMC2940775 Disease Relevance 0.13 Pain Relevance 0
However, as opposed to protamine, heparinase I did not produce any significant hemodynamic changes when given as a bolus to dogs.


Neg (not) Gene_expression (produce) of protamine
8) Confidence 0.00 Published 1996 Journal Anesthesiology Section Body Doc Link 8712450 Disease Relevance 0 Pain Relevance 0

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