INT63779

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Context Info
Confidence 0.64
First Reported 1995
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 6
Total Number 6
Disease Relevance 4.80
Pain Relevance 3.81

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Ado)
Anatomy Link Frequency
nerves 1
Duct 1
Ado (Rattus norvegicus)
Pain Link Frequency Relevance Heat
adenocard 63 100.00 Very High Very High Very High
Inflammation 17 99.48 Very High Very High Very High
tetrodotoxin 6 97.44 Very High Very High Very High
substance P 1 96.84 Very High Very High Very High
Action potential 1 96.08 Very High Very High Very High
Antinociceptive 2 95.20 Very High Very High Very High
Neurotransmitter 2 95.20 Very High Very High Very High
Peripheral nervous system 2 93.96 High High
anticonvulsant 1 92.16 High High
Spinal cord 2 87.56 High High
Disease Link Frequency Relevance Heat
Seborrhea 11 100.00 Very High Very High Very High
Patent Ductus Arteriosus 17 99.90 Very High Very High Very High
Hypoxia 15 99.82 Very High Very High Very High
INFLAMMATION 14 99.48 Very High Very High Very High
Down Syndrome 1 99.00 Very High Very High Very High
Nociception 3 98.08 Very High Very High Very High
Hypercapnia 2 95.88 Very High Very High Very High
Pulmonary Valve Stenosis 1 95.04 Very High Very High Very High
Cv General 2 Under Development 1 93.84 High High
Ventricular Heart Septal Defects 1 92.72 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
ADO is also released at sites of inflammation and it exerts anti-inflammatory effects via multiple mechanisms involving several cell types.
Localization (released) of ADO associated with adenocard and inflammation
1) Confidence 0.64 Published 1998 Journal Expert Opin Investig Drugs Section Abstract Doc Link 15991991 Disease Relevance 1.12 Pain Relevance 1.26
The results imply that severe hypoxia-induced release of NO and ADO, and the accompanying pial arteriolar dilation, are wholly dependent on the capacity to generate action potentials in perivascular nerves.
Localization (release) of ADO in nerves associated with action potential, adenocard and hypoxia
2) Confidence 0.48 Published 1995 Journal Brain Res. Section Abstract Doc Link 8750962 Disease Relevance 1.08 Pain Relevance 0.60
Inhibition of AK produces marked increases in extracellular ADO levels that are localized to cells and tissues undergoing accelerated ADO release.
Localization (release) of ADO associated with adenocard
3) Confidence 0.47 Published 1998 Journal Curr. Pharm. Des. Section Abstract Doc Link 10197052 Disease Relevance 0.25 Pain Relevance 0.61
AK inhibition selectively amplifies extracellular ADO levels at cell and tissue sites where accelerated release of ADO occurs.
Localization (release) of ADO associated with adenocard
4) Confidence 0.35 Published 2004 Journal Curr. Pharm. Des. Section Abstract Doc Link 15078144 Disease Relevance 0.84 Pain Relevance 0.80
Inhibition of AK potentiates local extracellular ADO levels at cell and tissue sites which are undergoing accelerated ADO release.
Localization (release) of ADO associated with adenocard
5) Confidence 0.32 Published 2000 Journal Expert Opin Investig Drugs Section Abstract Doc Link 11060695 Disease Relevance 0.32 Pain Relevance 0.54
For TCO of PDA, we used a SBD (Custom Medical Devices, Athens, Greece; TX, USA), an umbrella (USCI, Billerica, MA, USA), coils such as a Gianturco coil (Cook, Inc., Bloomington, IN, USA), a Duct-occlud device (PFM, Cologne, Germany), and an ADO (AGA, MN, USA).
Localization (used) of ADO in Duct associated with patent ductus arteriosus and seborrhea
6) Confidence 0.11 Published 2010 Journal Korean Circulation Journal Section Body Doc Link PMC2877787 Disease Relevance 1.19 Pain Relevance 0

General Comments

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