INT64113
From wiki-pain
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Sentences Mentioned In
Key: | Protein | Mutation | Event | Anatomy | Negation | Speculation | Pain term | Disease term |
CONCLUSION: Fluoxetine, an antidepressant, renormalizes dendrite atrophy of hippocampal neurons by inhibiting nitric oxide synthase overexpression in rat chronic mild stress model.
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Penile NOS content may be pharmacologically increased by: d) induction of NOS expression, and e) gene therapy with NOS cDNA. | |||||||||||||||
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Many investigators have reported increased expression of endothelial NOS in the glandular endometrium in patients with endometriosis [28,130]. | |||||||||||||||
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Expression of NOS is elevated in patients with endometriosis, and a common polymorphism of exon 7 at nucleotide 894 in the endothelial NOS gene may be associated with endometriosis [135]. | |||||||||||||||
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Peritoneal fluid NO levels, peritoneal macrophage NOS activity, and peritoneal macrophage inducible NOS protein expression has been examined in women with endometriosis-associated infertility. | |||||||||||||||
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Similarly, increased expression of nNOS (De Oliviera et al 2000) and iNOS (Madrigal et al 2001, 2003) has been noted to occur in limbic brain regions following acute restraint stress in rats. | |||||||||||||||
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GLNVA also suppressed accumulation of iROS and up-regulated heme oxygenase-1 expression. 6-OHDA-induced overexpression of nNOS, iNOS, COX-2, and gp91(phox) was also reduced by GLNVA. | |||||||||||||||
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GLNVA also suppressed accumulation of iROS and up-regulated heme oxygenase-1 expression. 6-OHDA-induced overexpression of nNOS, iNOS, COX-2, and gp91(phox) was also reduced by GLNVA. | |||||||||||||||
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GLNVA also decreased LPS activated microglia induced overexpression of neuronal nitric-oxide synthase (nNOS) and glycoprotein 91 phagocyte oxidase (gp91(phox)) on SH-SY5Y cells. | |||||||||||||||
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GLNVA also decreased LPS activated microglia induced overexpression of neuronal nitric-oxide synthase (nNOS) and glycoprotein 91 phagocyte oxidase (gp91(phox)) on SH-SY5Y cells. | |||||||||||||||
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GLNVA also decreased LPS activated microglia induced overexpression of neuronal nitric-oxide synthase (nNOS) and glycoprotein 91 phagocyte oxidase (gp91(phox)) on SH-SY5Y cells. | |||||||||||||||
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GLNVA also decreased LPS activated microglia induced overexpression of neuronal nitric-oxide synthase (nNOS) and glycoprotein 91 phagocyte oxidase (gp91(phox)) on SH-SY5Y cells. | |||||||||||||||
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The availability of selective nNOS inhibitors, as well as studies in gene-modified mice with perturbation of nNOS expression, have provided evidence that nonneuronal nNOS is physiologically active and exerts important regulatory influences in a number of different tissues. | |||||||||||||||
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Taken together, these findings suggest that NO derived from nNOS may have an important role in maintaining blood flow in the exercising skeletal muscle by reducing ? | |||||||||||||||
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In this context, experimental evidence revealed that increased activity and expression of nNOS may occur earlier than four hours in the paraventricular nucleus of rats subjected to lipopolysaccharide injection [27]. | |||||||||||||||
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In heart failure low NO levels are found proportionally to the severity of disease: the expression of No is depressed, but cytokine mediated i NOS expression is enhanced, causing exaggerated amounts of NO and producing myocyte damage or death. | |||||||||||||||
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In heart failure low NO levels are found proportionally to the severity of disease: the expression of No is depressed, but cytokine mediated i NOS expression is enhanced, causing exaggerated amounts of NO and producing myocyte damage or death. | |||||||||||||||
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These results suggest that inducible NOS is constitutively expressed in human duodenal enterocytes, is increased in patients with untreated celiac disease, and is partially corrected when such patients are treated. | |||||||||||||||
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CONCLUSION: These results suggest that the expression of NOS mainly contributes to the allergic symptoms. | |||||||||||||||
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Additional experiments demonstrated that acupuncture increased the level of iNOS mRNA in macrophages [44] and while examining the distributions of NO in the skin points of rats it was shown that NO contents and nNOS expression were consistently higher in the skin acupoints/meridians associated with low electric resistance [45]. | |||||||||||||||
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General Comments
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