INT64757

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Context Info
Confidence 0.03
First Reported 1996
Last Reported 1996
Negated 1
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.47

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

isomerase activity (Gpi1) carbohydrate metabolic process (Gpi1) cytoplasm (Gpi1)
cytosol (Gpi1) extracellular space (Gpi1) extracellular region (Gpi1)
oh (Mus musculus)
Gpi1 (Mus musculus)
Pain Link Frequency Relevance Heat
gABA 4 99.34 Very High Very High Very High
Catecholamine 3 81.52 Quite High
Opioid 3 77.80 Quite High
antagonist 3 72.32 Quite High

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Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The failure of diazepam to suppress NK activity also suggests that increased chloride flux through diazepam-sensitive GABA-A receptors (which is caused by EtOH as well as diazepam) does not mediate NK suppression in this model.
EtOH Neg (not) Positive_regulation (mediate) of Negative_regulation (suppression) of NK associated with gaba
1) Confidence 0.03 Published 1996 Journal J. Pharmacol. Exp. Ther. Section Abstract Doc Link 8819520 Disease Relevance 0 Pain Relevance 0.47

General Comments

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