INT66076

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Context Info
Confidence 0.00
First Reported 1996
Last Reported 2002
Negated 0
Speculated 0
Reported most in Abstract
Documents 3
Total Number 3
Disease Relevance 3.34
Pain Relevance 1.79

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Etax2 (Mus musculus)
Pain Link Frequency Relevance Heat
Migraine 25 99.90 Very High Very High Very High
potassium channel 1 99.58 Very High Very High Very High
Calcium channel 1 98.28 Very High Very High Very High
dopamine receptor 1 58.96 Quite High
Serotonin 1 48.08 Quite Low
Disease Link Frequency Relevance Heat
Ataxia 11 100.00 Very High Very High Very High
Migraine With Aura 13 99.90 Very High Very High Very High
Stress 1 95.12 Very High Very High Very High
Spinocerebellar Ataxia Type 2 1 94.88 High High
Vertigo 6 93.88 High High
Channelopathies 3 92.48 High High
Headache 12 88.12 High High
Syndrome 3 78.96 Quite High
Epilepsy 2 72.40 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Episodic ataxia type 1 (EA-1) is caused by missense mutations in the potassium channel gene KCNA1, whereas episodic ataxia type 2 (EA-2) is caused by missense and nonsense mutations in the calcium channel gene CACNA1A.
Positive_regulation (caused) of episodic ataxia type 2 associated with ataxia, calcium channel and potassium channel
1) Confidence 0.00 Published 2002 Journal Ann. N. Y. Acad. Sci. Section Abstract Doc Link 11960817 Disease Relevance 1.47 Pain Relevance 0.28
Familial hemiplegic migraine and episodic ataxia type-2 are caused by mutations in the Ca2+ channel gene CACNL1A4.
Positive_regulation (caused) of episodic ataxia type-2 associated with ataxia, migraine with aura and migraine
2) Confidence 0.00 Published 1996 Journal Cell Section Title Doc Link 8898206 Disease Relevance 0.80 Pain Relevance 0.67
Other mutation types cause episodic ataxia 2 (EA-2).
Positive_regulation (cause) of episodic ataxia 2 associated with ataxia
3) Confidence 0.00 Published 2001 Journal Ugeskr. Laeg. Section Abstract Doc Link 11723684 Disease Relevance 1.08 Pain Relevance 0.84

General Comments

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