INT66488

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Context Info
Confidence 0.01
First Reported 1996
Last Reported 1996
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 1.17

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Oprl1) plasma membrane (Oprl1) signal transducer activity (Oprl1)
Oprl1 (Mus musculus)
Cbm4 (Mus musculus)
Pain Link Frequency Relevance Heat
Enkephalin 3 97.56 Very High Very High Very High
tail-flick 3 96.88 Very High Very High Very High
agonist 3 92.08 High High
antinociception 9 88.08 High High
Opioid 3 79.24 Quite High
antagonist 4 75.00 Quite High
MU agonist 1 75.00 Quite High
mu opioid receptor 2 70.12 Quite High
opioid receptor 1 64.04 Quite High
Antinociceptive 1 50.72 Quite High

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Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Scatchard analysis of saturation binding data showed that N-CBM-TAMO decreased the Bmax value without affecting the Kd value for [3H][D-Ala2,(Me)Phe4, Gly(ol)5]enkephalin binding, whereas, N-CBM-TAMO increased the Kd value without altering the Bmax value for [3H]U69,593, which suggests that N-CBM-TAMO interacted covalently with the mu but not the kappa receptor.
kappa receptor Binding (interacted) of N-CBM-TAMO associated with enkephalin
1) Confidence 0.01 Published 1996 Journal J. Pharmacol. Exp. Ther. Section Abstract Doc Link 8930155 Disease Relevance 0 Pain Relevance 1.17

General Comments

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