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Context Info
Confidence 0.26
First Reported 1996
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.31
Pain Relevance 0.25

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

lipid binding (Apoa2) protein folding (Apoa2) transport (Apoa2)
extracellular space (Apoa2) extracellular region (Apoa2) cellular_component (Apoa2)
Anatomy Link Frequency
cardiovascular system 2
Apoa2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
anesthesia 1 94.48 High High
Kinase C 10 88.36 High High
Bioavailability 5 62.68 Quite High
cva 3 57.04 Quite High
aspirin 1 54.68 Quite High
antagonist 7 36.24 Quite Low
Inflammation 5 5.00 Very Low Very Low Very Low
Migraine 2 5.00 Very Low Very Low Very Low
Calcium channel 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Injury 6 100.00 Very High Very High Very High
Stress 15 96.60 Very High Very High Very High
Increased Venous Pressure Under Development 27 87.88 High High
Renal Disease 4 81.92 Quite High
Hyperglycemia 9 70.88 Quite High
Aging 6 69.68 Quite High
Pressure And Volume Under Development 18 69.24 Quite High
Hyperlipidemia 11 68.68 Quite High
Diabetes Mellitus 29 67.80 Quite High
Cv General 3 Under Development 3 57.04 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The levels of 125I-Sar1.Ile8-AII bound to membrane preparations of the wound tissues decreased at early time points (from 0.5 to 4 h), increased from day 1 to day 7, and returned to nonsurgical levels by day 10.
Positive_regulation (increased) of Ile8-AII Binding (bound) of associated with injury
1) Confidence 0.26 Published 1996 Journal J Invest Surg Section Abstract Doc Link 8981218 Disease Relevance 0.53 Pain Relevance 0.09
In these effects an activated ET is likely to mediate the activity of the AII through NOX activation as AII-evoked expression of ET-1 in adventitial fibroblasts appears to be mediated, at least in part, by NOX.66 The mitochondrial ROS is responsible to mediate the activity of ET-1 in the cardiovascular system.67 The vascular activity of AII is closely related to ET-1, coronary vasoconstriction by ?
Positive_regulation (mediate) of AII Binding (activity) of in cardiovascular system associated with increased venous pressure under development
2) Confidence 0.03 Published 2010 Journal Vascular Health and Risk Management Section Body Doc Link PMC2941789 Disease Relevance 0.78 Pain Relevance 0.16

General Comments

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