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Context Info
Confidence 0.54
First Reported 1997
Last Reported 1997
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.06
Pain Relevance 0.43

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Scn10a) plasma membrane (Scn10a) transmembrane transport (Scn10a)
Scn10a (Mus musculus)
Pain Link Frequency Relevance Heat
sodium channel 6 96.32 Very High Very High Very High
tetrodotoxin 3 75.00 Quite High
Spinal cord 1 57.16 Quite High
unmyelinated 1 49.88 Quite Low
Disease Link Frequency Relevance Heat
Nociception 1 55.92 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These results provide the basis for an evolutionary comparison of sodium channels, the construction and analysis of a mouse SNS null mutant as a direct approach to understanding the biological function of SNS, and the identification of regulatory elements that are responsible for the tissue- and cell-specific expression of SNS.
Regulation (responsible) of Gene_expression (expression) of SNS associated with sodium channel
1) Confidence 0.54 Published 1997 Journal Genomics Section Abstract Doc Link 9143495 Disease Relevance 0.06 Pain Relevance 0.43

General Comments

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