INT69591

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Context Info
Confidence 0.50
First Reported 1997
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 3
Disease Relevance 3.06
Pain Relevance 3.99

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

enzyme regulator activity (PSMD1) cytosol (PSMD1) nucleoplasm (PSMD1)
small molecule metabolic process (PSMD1) cellular nitrogen compound metabolic process (PSMD1)
Anatomy Link Frequency
insular cortex 3
digits 2
prefrontal 1
PSMD1 (Homo sapiens)
Pain Link Frequency Relevance Heat
primary somatosensory cortex 13 100.00 Very High Very High Very High
Anterior cingulate cortex 4 100.00 Very High Very High Very High
amygdala 2 100.00 Very High Very High Very High
Thalamus 2 100.00 Very High Very High Very High
allodynia 22 97.92 Very High Very High Very High
Pain 26 82.80 Quite High
central pain 2 79.68 Quite High
Somatosensory cortex 1 75.00 Quite High
imagery 2 70.16 Quite High
positron emission tomography 1 67.72 Quite High
Disease Link Frequency Relevance Heat
Neuropathic Pain 22 97.92 Very High Very High Very High
Amputation 4 91.60 High High
Pain 20 81.12 Quite High
Paralysis 1 57.44 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The area of S1 activated during VS of the digits in a normal control subject was coextensive with the ventral S1 region abnormally activated during VS of the ectopic phantom representation in the two amputees, suggesting that the deafferented digit or hand/arm area had been activated by sensory input from the pectoral region.
Positive_regulation (activated) of Positive_regulation (activated) of S1 in digits associated with primary somatosensory cortex
1) Confidence 0.50 Published 1997 Journal J. Neurophysiol. Section Abstract Doc Link 9163390 Disease Relevance 0.34 Pain Relevance 0.64
Subgroup analysis using rating-weighted predictors revealed activation of the contralateral thalamus, anterior cingulate cortex, and amygdala and a bilateral activation of S1, S2, and insular cortex and prefrontal cortices in allodynia-experienced subjects.
Positive_regulation (activation) of Positive_regulation (activation) of S1 in insular cortex associated with allodynia, thalamus, amygdala and anterior cingulate cortex
2) Confidence 0.35 Published 2008 Journal J Pain Section Abstract Doc Link 18455481 Disease Relevance 1.36 Pain Relevance 1.68
Subgroup analysis using rating-weighted predictors revealed activation of the contralateral thalamus, anterior cingulate cortex, and amygdala and a bilateral activation of S1, S2, and insular cortex and prefrontal cortices in allodynia-experienced subjects.
Positive_regulation (activation) of in prefrontal Positive_regulation (activation) of S1 in insular cortex associated with allodynia, thalamus, amygdala and anterior cingulate cortex
3) Confidence 0.12 Published 2008 Journal J Pain Section Abstract Doc Link 18455481 Disease Relevance 1.36 Pain Relevance 1.68

General Comments

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