INT70090

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Context Info
Confidence 0.03
First Reported 1997
Last Reported 1997
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.59
Pain Relevance 0.15

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nuclear envelope (Ache) Golgi apparatus (Ache) transferase activity, transferring acyl groups (TGM1)
cytoplasm (Ache) protein modification process (TGM1) extracellular space (Ache)
TGM1 (Homo sapiens)
Ache (Rattus norvegicus)
Pain Link Frequency Relevance Heat
monoamine 1 100.00 Very High Very High Very High
cINOD 1 100.00 Very High Very High Very High
Disease Link Frequency Relevance Heat
INFLAMMATION 1 100.00 Very High Very High Very High
Disease 4 96.00 Very High Very High Very High
Alzheimer's Dementia 2 83.20 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This report evaluated the potential of a total of ten different pharmacological agents to inhibit TGase-induced cross-linking of A beta, including known TGase inhibitors (dansylcadaverine, spermine), non-steroidal anti-inflammatory drugs (indomethacin, meclofenamic acid, diflunisal, salicylic acid), monoamine oxidase inhibitors (tranylcypromine, phenelzine), an acetylcholinesterase inhibitor (tacrine), and an iron chelating agent (deferoxamine).
TGase-induced Positive_regulation (induced) of acetylcholinesterase Binding (cross-linking) of associated with inflammation, cinod and monoamine
1) Confidence 0.03 Published 1997 Journal Life Sci. Section Abstract Doc Link 9194688 Disease Relevance 0.59 Pain Relevance 0.15

General Comments

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