INT70302

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Context Info
Confidence 0.66
First Reported 1997
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 3
Total Number 3
Disease Relevance 0.24
Pain Relevance 0.44

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (NEFH) cell death (NEFH) molecular_function (NEFH)
Anatomy Link Frequency
neuronal 1
neurons 1
spinal cord neurons 1
NEFH (Homo sapiens)
Pain Link Frequency Relevance Heat
Spinal cord 6 98.16 Very High Very High Very High
adenocard 1 85.84 High High
induced neuropathy 2 65.56 Quite High
tetrodotoxin 12 5.00 Very Low Very Low Very Low
sodium channel 4 5.00 Very Low Very Low Very Low
potassium channel 4 5.00 Very Low Very Low Very Low
Calcium channel 3 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Neuropathic Pain 2 65.56 Quite High
Disease 7 61.12 Quite High
Obesity 19 50.00 Quite Low
Motor Neuron Diseases 2 47.16 Quite Low
Neurodegenerative Disease 5 43.72 Quite Low
Injury 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Middle and high NF subunit (NFH) mRNA levels were not significantly different from polyadenylated mRNA levels in spinal cord neurons in aluminum-treated/control animals.
Transcription (levels) of NFH in spinal cord neurons associated with spinal cord
1) Confidence 0.66 Published 1997 Journal Neurotoxicology Section Abstract Doc Link 9215990 Disease Relevance 0.13 Pain Relevance 0.20
NFH mRNA levels were decreased in neurons containing aluminum-induced NF accumulations.
Transcription (levels) of NFH in neurons
2) Confidence 0.49 Published 1997 Journal Neurotoxicology Section Abstract Doc Link 9215990 Disease Relevance 0.12 Pain Relevance 0.19
Our experiments demonstrate that NI-hADSCs express increased immunoreactivities for neuronal markers Tuj1, MAP2, NFL, NFM, NFH, NSE, NeuN, GAP43, and SNAP25, as well as the increased mRNA expression of Tuj1, MAP2, NFL, NFM, NSE, GAP43, and SNAP25 compared to primary hADSCs (Figure 3), indicating that hADSCs differentiate into neural cells via bFGF and forskolin-mediated differentiation.
Transcription (expression) of NFH in neuronal
3) Confidence 0.39 Published 2010 Journal BMC Cell Biol Section Body Doc Link PMC2867791 Disease Relevance 0 Pain Relevance 0.04

General Comments

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