INT71329

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Context Info
Confidence 0.01
First Reported 1997
Last Reported 1997
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.71
Pain Relevance 0.15

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell death (Cacna1a) plasma membrane (Cacna1a) nucleus (Cacna1a)
neurological system process (Cacna1a) transmembrane transport (Cacna1a) cytoplasm (Cacna1a)
Cacna1a (Rattus norvegicus)
Ea2 (Mus musculus)
Pain Link Frequency Relevance Heat
Migraine 1 100.00 Very High Very High Very High
Calcium channel 1 100.00 Very High Very High Very High
Disease Link Frequency Relevance Heat
Ataxia 5 100.00 Very High Very High Very High
Migraine With Aura 1 100.00 Very High Very High Very High
Spinocerebellar Ataxia Type 2 5 85.36 High High
Ocular Toxicity (including Many Sub-types) 1 61.68 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Point mutations of the CACNA1A gene coding for the alpha 1A voltage-dependent calcium channel subunit are responsible for familial hemiplegic migraine (FHM) and episodic ataxia type 2 (EA2).
CACNA1A gene Regulation (responsible) of EA2 associated with ataxia, migraine with aura, migraine and calcium channel
1) Confidence 0.01 Published 1997 Journal Hum. Mol. Genet. Section Abstract Doc Link 9302278 Disease Relevance 0.71 Pain Relevance 0.15

General Comments

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