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Context Info
Confidence 0.32
First Reported 1997
Last Reported 1997
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.89
Pain Relevance 0.96

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Pars2) ligase activity (Pars2) cytoplasm (Pars2)
Anatomy Link Frequency
neurons 2
Pars2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Glutamate 3 98.72 Very High Very High Very High
Eae 8 95.16 Very High Very High Very High
Neuropathic pain 7 93.76 High High
Sciatic nerve 2 93.64 High High
Dorsal horn neuron 3 85.44 High High
Spinal cord 4 84.64 Quite High
Dorsal horn 2 77.60 Quite High
intrathecal 2 58.32 Quite High
Thermal hyperalgesia 1 44.16 Quite Low
Hyperalgesia 1 41.52 Quite Low
Disease Link Frequency Relevance Heat
Drug Induced Neurotoxicity 3 98.08 Very High Very High Very High
Injury 8 95.16 Very High Very High Very High
Neuropathic Pain 8 93.76 High High
Hyperalgesia 2 44.16 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In the present study, we examined whether the inhibition of the nitric oxide (NO)-activated poly(ADP-ribose) synthetase (PARS), a nuclear enzyme critical to glutamate-induced neurotoxicity, would both reduce the incidence of dark neurons and attenuate behavioral manifestations of neuropathic pain in CCI rats.
Spec (whether) Negative_regulation (inhibition) of Spec (whether) Positive_regulation (-activated) of PARS in neurons associated with glutamate, drug induced neurotoxicity, eae and neuropathic pain
1) Confidence 0.32 Published 1997 Journal Pain Section Abstract Doc Link 9313276 Disease Relevance 0.89 Pain Relevance 0.96

General Comments

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