INT72041

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Context Info
Confidence 0.46
First Reported 1997
Last Reported 2008
Negated 1
Speculated 0
Reported most in Body
Documents 4
Total Number 4
Disease Relevance 0.74
Pain Relevance 0.71

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
proximal 2
nervous system 2
hippocampus 2
L1cam (Mus musculus)
Pain Link Frequency Relevance Heat
Hippocampus 16 99.68 Very High Very High Very High
substance P 12 93.92 High High
Neuropeptide 4 92.60 High High
Spinal cord 12 86.36 High High
cerebral cortex 15 84.64 Quite High
amygdala 22 83.16 Quite High
Central nervous system 12 79.84 Quite High
Pain 1 73.56 Quite High
Substantia nigra 22 60.00 Quite High
imagery 32 57.48 Quite High
Disease Link Frequency Relevance Heat
Anxiety Disorder 8 99.72 Very High Very High Very High
Adhesions 3 98.20 Very High Very High Very High
Intellectual Impairment 1 87.00 High High
Paraplegia 1 81.00 Quite High
Hydrocephalus 1 80.04 Quite High
Pain 1 73.56 Quite High
Fasciculation 1 59.32 Quite High
Urological Neuroanatomy 14 5.00 Very Low Very Low Very Low
Nociception 5 5.00 Very Low Very Low Very Low
Heart Rate Under Development 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Behavioral analysis has shown that contextual fear conditioning induced L1 expression in the hippocampus [13].
Positive_regulation (induced) of Gene_expression (expression) of L1 in hippocampus associated with anxiety disorder and hippocampus
1) Confidence 0.46 Published 2003 Journal BMC Neurosci Section Body Doc Link PMC155647 Disease Relevance 0.26 Pain Relevance 0.33
Disruption of the mouse L1 gene leads to malformations of the nervous system.
Positive_regulation (leads) of Gene_expression (Disruption) of L1 in nervous system
2) Confidence 0.43 Published 1997 Journal Nat. Genet. Section Title Doc Link 9354804 Disease Relevance 0.48 Pain Relevance 0.07
It was found that there was no accumulation of L1 on the proximal side of the transected nerve (Fig. 2a,2a'), and no reduction of L1 on the distal side of the lesion (Fig. 2c,2c') or in the terminal field (data not shown).
Neg (no) Positive_regulation (accumulation) of Neg (no) Gene_expression (reduction) of L1 in proximal
3) Confidence 0.43 Published 2003 Journal BMC Neurosci Section Body Doc Link PMC155647 Disease Relevance 0 Pain Relevance 0.26
14s and 22s for L1 and 10?
Positive_regulation (22s) of Gene_expression (14s) of L1
4) Confidence 0.06 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2424136 Disease Relevance 0 Pain Relevance 0.05

General Comments

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