INT72251

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Context Info
Confidence 0.05
First Reported 1997
Last Reported 2004
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.07
Pain Relevance 0.13

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endoplasmic reticulum (Kcnma1) plasma membrane (Kcnma1)
Anatomy Link Frequency
myocytes 1
brain 1
Kcnma1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ASIC 2 93.52 High High
dorsal root ganglion 2 75.00 Quite High
tetrodotoxin 1 50.00 Quite Low
Pain 1 28.80 Quite Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 2 75.00 Quite High
Myocardial Infarction 2 50.00 Quite Low
Pain 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
CONCLUSIONS: Our data suggest that I(Ca(TTX))-like current in PMI myocytes does not arise from classical Na(+) channels modified by oxidative stress or PKA phosphorylation and most probably represents a different Na(+) channel type re-expressed in some cells after remodelling.


Phosphorylation (phosphorylation) of channel in myocytes
1) Confidence 0.05 Published 2004 Journal Cardiovasc. Res. Section Body Doc Link 15306221 Disease Relevance 0 Pain Relevance 0
In addition, we have cloned from mouse and rat brain a splice variant form of the MDEG1 channel which differs in the first 236 amino acids, including the first transmembrane region.
Phosphorylation (form) of channel in brain
2) Confidence 0.01 Published 1997 Journal J. Biol. Chem. Section Abstract Doc Link 9368048 Disease Relevance 0.07 Pain Relevance 0.13

General Comments

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