INT76844

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Context Info
Confidence 0.46
First Reported 1998
Last Reported 2010
Negated 3
Speculated 0
Reported most in Body
Documents 3
Total Number 8
Disease Relevance 0.84
Pain Relevance 0.89

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (Rbm19) nucleolus (Rbm19) chromosome (Rbm19)
RNA binding (Rbm19) nucleus (Rbm19) cytoplasm (Rbm19)
Anatomy Link Frequency
PGE2 1
Rbm19 (Mus musculus)
Pain Link Frequency Relevance Heat
qutenza 12 99.44 Very High Very High Very High
analgesia 1 99.40 Very High Very High Very High
antagonist 7 95.84 Very High Very High Very High
Chronic pancreatitis 1 75.00 Quite High
Analgesic 1 25.00 Low Low
imagery 18 5.00 Very Low Very Low Very Low
Pain 12 5.00 Very Low Very Low Very Low
cINOD 6 5.00 Very Low Very Low Very Low
addiction 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 60 92.32 High High
Pancreatitis 2 90.24 High High
Hypoxia 60 89.44 High High
Volume Depletion And Dehydration 66 89.08 High High
Reticulocytosis 6 86.08 High High
Anaemia 6 85.32 High High
Sickle Cell Anemia 24 70.84 Quite High
Stress 18 5.00 Very Low Very Low Very Low
Hemolytic Anemia 12 5.00 Very Low Very Low Very Low
Pain 12 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The mainstay of therapy includes aggressive hydration, maintenance of NPO, and adequate analgesia with narcotics.
Positive_regulation (maintenance) of NPO associated with analgesia
1) Confidence 0.46 Published 2001 Journal Prim. Care Section Abstract Doc Link 11483447 Disease Relevance 0.17 Pain Relevance 0.17
Exposure of the sensory neuron to either PGE2 or forskolin produced a large and transient increase in the mean channel activity (NPo) elicited by capsaicin, although the unitary conductance remained unaltered.
Positive_regulation (increase) of NPo in PGE2 associated with qutenza
2) Confidence 0.40 Published 1998 Journal J. Neurosci. Section Abstract Doc Link 9698303 Disease Relevance 0 Pain Relevance 0.72
The normoxic NPo of 0.0377±0.0375 in CO-pretreated cells did not increase upon deoxygenation (NPo?
Neg (not) Positive_regulation (increase) of NPo
3) Confidence 0.10 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2806905 Disease Relevance 0 Pain Relevance 0
As was found for deoxygenation-activated Ca2+ elevation, inclusion in the pipette of 1 µM GsMTx-4 prevented the deoxygenation-induced increase in cation conductance (NPo 0.003±0.003, n?
Positive_regulation (increase) of NPo
4) Confidence 0.10 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2806905 Disease Relevance 0.09 Pain Relevance 0
As shown in Figure 2B, deoxygenation increased mean NPo in SAD mouse red cell patches from 0.01±0.013 to 0.48±0.20 (n?
Positive_regulation (increased) of NPo
5) Confidence 0.06 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2806905 Disease Relevance 0 Pain Relevance 0
Deoxygenation increased SAD red cell patch NPo from the room air value of 0.01±0.01 to 0.65±0.22 (n?
Positive_regulation (increased) of NPo
6) Confidence 0.06 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2806905 Disease Relevance 0.50 Pain Relevance 0
The normoxic NPo of 0.0377±0.0375 in CO-pretreated cells did not increase upon deoxygenation (NPo?
Neg (not) Positive_regulation (increase) of NPo
7) Confidence 0.06 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2806905 Disease Relevance 0 Pain Relevance 0
Patch NPo was not different from zero in room air, but increased to 0.90±0.23 upon deoxygenation (P<0.05; n?
Neg (not) Positive_regulation (increased) of NPo
8) Confidence 0.06 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2806905 Disease Relevance 0.08 Pain Relevance 0

General Comments

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