INT77146
From wiki-pain
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Sentences Mentioned In
Key: | Protein | Mutation | Event | Anatomy | Negation | Speculation | Pain term | Disease term |
, interleukin-2 (IL-2), interleukin-4 (IL-4), interleukin-6 (IL-6), interleukin-8 (IL-8), interleukin-10 (IL-10), tumour necrosis factor-alpha (TNF-?) | |||||||||||||||
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The inhibitory effect of ASA on IL-4 transcription was not mediated by decreased nuclear expression of the known salicylate target nuclear factor (NF)-kappaB and was accompanied by reduced binding of an inducible factor to an IL-4 promoter region upstream of, but not overlapping, the NF of activated T cells- and NF-kappaB-binding P1 element. | |||||||||||||||
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IL-4 and other suppressive cytokines that can be produced by Th2 cells (e.g. | |||||||||||||||
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Although we show that IL-7, and in particular the combination of IL-7 and IL-4, increased Th2 activity rather than Th1 activity of naive CD4+ T cells, this was only found under conditions where IFN-? | |||||||||||||||
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The combination of IL-7 and IL-4 reduced IFN-? | |||||||||||||||
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The purpose of this study was to evaluate the levels of nitric oxide (NO), IL-4, TGF-beta, tumor necrosis factor-alpha (TNF-alpha), and interferon-gamma (IFN-gamma) in chronic periapical lesions and to determine their possible association with clinical and radiographic parameters. | |||||||||||||||
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The scatter plot of log signal intensities from the HV pool (horizontal) and patient TA8 (vertical) show that the signal intensities of IL-4 and IL-10 are considerably lower than that of DEFA3. | |||||||||||||||
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, IL-4, and IL-2 upon rechallenge in vitro with allergen [165]. | |||||||||||||||
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Levels tended to normalize to baseline values within 34 weeks of infection in all animals, with IL2, IL4, IL12p40, IFN? | |||||||||||||||
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At two weeks after the initial vaccination (on day 15), the 5 immunization sites were punch biopsied and evaluated to determine the dose of IL-4 associated with the most intense mononuclear cell infiltrate. | |||||||||||||||
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Interestingly, IL-4+TGF-? | |||||||||||||||
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alone or in combination with either IL-4 or TGF-? | |||||||||||||||
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We found that IL-4+TGF-? | |||||||||||||||
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Accordingly, the IL-4 : IFN-? | |||||||||||||||
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Activation of melatonin receptors enhances the release of T-helper cell type 1 (Th1) cytokines, such as gamma-interferon and interleukin-2, as well as of novel opioid cytokines which crossreact immunologically with both interleukin-4 and dynorphin B. | |||||||||||||||
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These include the release of IL-1, IL-2, IL-4, IL-6, IL-8, IL-10, tumor necrosis factor-alfa (TNF-alfa), platelet-activating factor, endorphins, nitric oxide, reactive oxygen species, tissue factor, macrophage migration inhibitory factor, and chemokines, which lead to a variety of immune system responses including vasodilatation, enhanced expression of adhesion molecules, increased capillary permeability, increased clot formation, and decreased fibrinolysis. | |||||||||||||||
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Some of the patients suffered also from atopic dermatitis (AD) and were exposed to cat and/or other pet allergens, associated with an increased IL-4 and decreased IFN-gamma production. | |||||||||||||||
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[42], anti-CD3/CD28 antibodies or antigen specific stimulation [43,44], IL-4/IL-13 [45,46], and thrombospondin-CD47 interaction [46]. | |||||||||||||||
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Activation of MLT receptors enhances the release of T-helper cell cytokines, such as gamma-interferon and interleukin-2 (IL-2), as well as activation of novel opioid cytokines which crossreact immunologically with both interleukin-4 and dynorphin B. | |||||||||||||||
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, are more likely to be associated with allograft rejection than Th2-type cytokines, such as IL-4, IL? | |||||||||||||||
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General Comments
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