INT78714

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Context Info
Confidence 0.50
First Reported 1998
Last Reported 2005
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.63
Pain Relevance 0.78

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Ntrk1) signal transduction (Ngf) extracellular space (Ngf)
extracellular region (Ngf) plasma membrane (Ntrk1) kinase activity (Ntrk1)
Ntrk1 (Mus musculus)
Ngf (Mus musculus)
Pain Link Frequency Relevance Heat
Nerve growth factor 7 100.00 Very High Very High Very High
Hyperalgesia 4 98.64 Very High Very High Very High
withdrawal 1 89.00 High High
Pain 1 67.92 Quite High
IPN 2 65.00 Quite High
Disease Link Frequency Relevance Heat
Hyperalgesia 4 98.64 Very High Very High Very High
Sprains And Strains 1 85.52 High High
Targeted Disruption 1 75.64 Quite High
Pain 1 67.92 Quite High
Inflammatory Pain 2 65.00 Quite High
Neuropathic Pain 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Detailed knowledge of the molecular basis determining the specificity of this antibody is of importance because of its potential use as a modulator of the TrkA-mediated NGF activity.
TrkA Positive_regulation (mediated) of NGF
1) Confidence 0.50 Published 2005 Journal Proteins Section Abstract Doc Link 15625712 Disease Relevance 0 Pain Relevance 0
This means that NGF-induced hyperalgesia can occur in the absence of the p75 receptor and suggests that the trkA receptor is sufficient to mediate the acute noxious action of NGF.
trkA receptor Positive_regulation (mediate) of NGF associated with hyperalgesia and nerve growth factor
2) Confidence 0.03 Published 1998 Journal Neurosci. Lett. Section Abstract Doc Link 9835221 Disease Relevance 0.63 Pain Relevance 0.78

General Comments

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