INT78972

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Context Info
Confidence 0.11
First Reported 1998
Last Reported 1998
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.09
Pain Relevance 0.82

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Ina) nucleoplasm (Prkaca) mitochondrion (Prkaca)
Golgi apparatus (Prkaca) protein complex (Prkaca) cytoplasm (Prkaca)
Anatomy Link Frequency
sensory neurons 1
Prkaca (Rattus norvegicus)
Ina (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Kinase C 22 99.12 Very High Very High Very High
tetrodotoxin 2 96.76 Very High Very High Very High
Action potential 2 92.24 High High
Disease Link Frequency Relevance Heat
Nociception 2 89.76 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Modulation of TTX-R INa by PKC and PKA and their role in PGE2-induced sensitization of rat sensory neurons in vitro.
PKA Regulation (Modulation) of TTX-R INa in sensory neurons
1) Confidence 0.11 Published 1998 Journal J. Neurosci. Section Title Doc Link 9852572 Disease Relevance 0.09 Pain Relevance 0.38
Thus, although TTX-R INa is a common target for cellular processes involving the activation of either PKA or PKC, PKC activity is necessary to enable subsequent PKA-mediated modulation of TTX-R INa.
PKA-mediated Regulation (modulation) of TTX-R INa associated with kinase c
2) Confidence 0.05 Published 1998 Journal J. Neurosci. Section Abstract Doc Link 9852572 Disease Relevance 0 Pain Relevance 0.43

General Comments

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