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Context Info
Confidence 0.13
First Reported 1998
Last Reported 1998
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.16
Pain Relevance 0.93

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Ina) intracellular (Prkcg) response to stress (Prkcg)
cytoplasm (Prkcg) cytosol (Prkcg) structural molecule activity (Ina)
Anatomy Link Frequency
sensory neurons 1
Prkcg (Rattus norvegicus)
Ina (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Kinase C 22 100.00 Very High Very High Very High
tetrodotoxin 2 96.44 Very High Very High Very High
Action potential 2 91.92 High High
Disease Link Frequency Relevance Heat
Nociception 2 89.44 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Modulation of TTX-R INa by PKC and PKA and their role in PGE2-induced sensitization of rat sensory neurons in vitro.
PKC Regulation (Modulation) of TTX-R INa in sensory neurons
1) Confidence 0.13 Published 1998 Journal J. Neurosci. Section Title Doc Link 9852572 Disease Relevance 0.09 Pain Relevance 0.38
Investigating the interaction between PKC and PKA revealed that although inhibitors of PKA had little effect on PKC-induced modulation of TTX-R INa, inhibitors of PKC significantly attenuated PKA-induced modulation of the current.
PKC-induced Regulation (modulation) of TTX-R INa associated with kinase c
2) Confidence 0.06 Published 1998 Journal J. Neurosci. Section Abstract Doc Link 9852572 Disease Relevance 0.07 Pain Relevance 0.55

General Comments

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