INT79442

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Context Info
Confidence 0.19
First Reported 1999
Last Reported 2004
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.95
Pain Relevance 1.89

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (XCL1) extracellular space (XCL1) extracellular region (XCL1)
cell-cell signaling (XCL1)
Anatomy Link Frequency
BEAS-2B 1
head 1
XCL1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 11 100.00 Very High Very High Very High
Pain 9 98.68 Very High Very High Very High
Glutamate 5 98.38 Very High Very High Very High
qutenza 5 88.28 High High
antagonist 3 87.72 High High
Inflammation 5 84.24 Quite High
substance P 3 80.16 Quite High
Neuropeptide 1 53.92 Quite High
Disease Link Frequency Relevance Heat
Pain 6 98.68 Very High Very High Very High
INFLAMMATION 5 84.24 Quite High
Neck Pain 1 79.92 Quite High
Myalgia 2 62.44 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Glutamate-evoked SP pain was associated with an increase in SP EMG activity at rest and a decrease in SCM EMG activity in the head right position.
Negative_regulation (decrease) of SCM EMG in head associated with pain and glutamate
1) Confidence 0.19 Published 2004 Journal Pain Section Abstract Doc Link 15157682 Disease Relevance 0.66 Pain Relevance 0.94
BEAS-2B cells exposed to ROFA in calcium-free media failed to demonstrate increases of [Ca2+]i and showed reduced levels of cytokine transcript (i.e., IL-6, IL-8, TNFalpha) and IL-6 release, suggesting that ROFA-stimulated cytokine formation was partially dependent on extracellular calcium sources.
Negative_regulation (reduced) of cytokine transcript in BEAS-2B associated with cytokine
2) Confidence 0.00 Published 1999 Journal Toxicol. Appl. Pharmacol. Section Abstract Doc Link 9882597 Disease Relevance 0.29 Pain Relevance 0.95

General Comments

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