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Context Info
Confidence 0.00
First Reported 1999
Last Reported 1999
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 1.20

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Akr1d1) mitochondrion (Abat) oxidoreductase activity (Akr1d1)
cytoplasm (Akr1d1)
Anatomy Link Frequency
neuronal 2
Akr1d1 (Rattus norvegicus)
Abat (Rattus norvegicus)
Pain Link Frequency Relevance Heat
gABA 11 100.00 Very High Very High Very High
Substantia nigra 6 98.88 Very High Very High Very High
Dopamine 2 96.00 Very High Very High Very High
agonist 4 91.60 High High
antagonist 1 88.76 High High
tetrodotoxin 3 71.36 Quite High
dopamine receptor 1 55.12 Quite High


Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These data indicate that although basal extracellular concentrations of GABA in the substantia nigra may not be derived from neuronal pools, K+ -stimulated release of GABA is impulse-mediated and is modulated by the D1 and the D2 receptors.
D1 Regulation (modulated) of Localization (release) of GABA in neuronal associated with gaba and substantia nigra
1) Confidence 0.00 Published 1999 Journal Synapse Section Abstract Doc Link 10188635 Disease Relevance 0 Pain Relevance 1.20

General Comments

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