INT81185

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Context Info
Confidence 0.11
First Reported 1999
Last Reported 1999
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.74
Pain Relevance 0.77

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signal transduction (Vipr2) plasma membrane (Vipr2) signal transducer activity (Vipr2)
Anatomy Link Frequency
dorsal horn 2
Vipr2 (Mus musculus)
Pain Link Frequency Relevance Heat
Dorsal horn 2 98.48 Very High Very High Very High
agonist 3 95.44 Very High Very High Very High
Eae 4 91.76 High High
antagonist 2 86.24 High High
Neuropathic pain 1 79.92 Quite High
spinal dorsal horn 1 72.40 Quite High
Peripheral nerve injury 1 68.12 Quite High
dorsal root ganglion 1 66.52 Quite High
Analgesic 1 60.48 Quite High
Lasting pain 1 58.80 Quite High
Disease Link Frequency Relevance Heat
Neuropathic Pain 2 97.60 Very High Very High Very High
Injury 4 91.76 High High
Diabetic Nephropathy 1 75.00 Quite High
Nervous System Injury 2 68.12 Quite High
Ganglion Cysts 1 66.52 Quite High
Pain 1 58.80 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In situ hybridisation histochemistry (ISHH) confirmed an increase in the expression of VPAC2 receptor mRNA within the ipsilateral dorsal horn following neuropathy, while VPAC1 receptor mRNA was seen to decrease and that for PAC1 receptors remained unchanged.
Positive_regulation (increase) of Transcription (expression) of VPAC2 in dorsal horn associated with neuropathic pain and dorsal horn
1) Confidence 0.11 Published 1999 Journal Neuropharmacology Section Abstract Doc Link 10193908 Disease Relevance 0.74 Pain Relevance 0.77

General Comments

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