INT81264
From wiki-pain
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Sentences Mentioned In
Key: | Protein | Mutation | Event | Anatomy | Negation | Speculation | Pain term | Disease term |
The results of these studies indicated that substance P and neurokinin A can directly induce nerve growth factor mRNA expression and the secretion of bioactive nerve growth factor protein in both human and murine keratinocytes. | |||||||||||||||
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Neutralization of nerve growth factor induces plasticity of ATP-sensitive P2X3 receptors of nociceptive trigeminal ganglion neurons. | |||||||||||||||
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In this study, we didn't find any enhanced motility of microglia after local application of MCP-1, FKN, BDNF, NGF, IL-1? | |||||||||||||||
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The secretion of glia cell-derived neurotrophic factor (GDNF), nerve growth factor (NGF) and transforming growth factor-beta (TGF-beta) contributes to the maintenance of epithelial integrity and the secretion of endothelins might be involved in vasoregulation. | |||||||||||||||
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Therefore, it is reasonable to propose that NGF secreted by melanoma tumor was transferred through blood and bound to its cognate receptors in the salivary gland, ultimately resulting in the stimulation of multiple TFs expression including Egr-1 (Fig. 6). | |||||||||||||||
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Prior studies showed that DRG explants from Runx3-knockout mouse embryos extended short neurites in the presence of NT3, a ligand for TrkC, but not in the presence of NGF, a ligand for TrkA [16]. | |||||||||||||||
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Inflammation can lead to an enhanced production and release of NGF. | |||||||||||||||
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Inflammation can lead to an enhanced production and release of NGF. | |||||||||||||||
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RSV-induced release of NGF may lead to short- and long-term changes in the distribution and reactivity of sensory nerves across the respiratory tract, thus participating in exaggerated inflammatory reactions during and after the infection. | |||||||||||||||
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The experimental model was the mouse PC12 neuroblastoma cell line which can be induced to differentiate into neurons in the presence of NGF (nerve growth factor). | |||||||||||||||
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The experimental model was the mouse PC12 neuroblastoma cell line which can be induced to differentiate into neurons in the presence of NGF (nerve growth factor). | |||||||||||||||
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Pro-inflammatory agents such as nerve growth factor (NGF), ATP, bradykinins, serotonin, histamine, proteases and chemokines lead to TRPV1 sensitization through phospholipase C activation. | |||||||||||||||
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Pro-inflammatory agents such as nerve growth factor (NGF), ATP, bradykinins, serotonin, histamine, proteases and chemokines lead to TRPV1 sensitization through phospholipase C activation. | |||||||||||||||
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After local tissue injury, the release of chemical mediators such as potassium ions, ATP, bradykinin and prostaglandin E2 directly activate nerve endings and indirectly trigger the release of algesic mediators such as histamine, 5-hydroxytryptamine and nerve growth factor from other cells, which, in turn, stimulate proximal afferent nerve endings and silent nociceptors. | |||||||||||||||
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The relevance of trophic support, mainly NGF and IGF-1, and other growth factor signaling through trk receptors and activation of PI3K/Akt and PKC, is also linked to the key role played by GSK-3? | |||||||||||||||
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An increase in TRPV1 expression occurs in primary sensory neurons after peripheral inflammation and requires retrograde transport of nerve growth factor (NGF). | |||||||||||||||
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General Comments
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