INT82240

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Context Info
Confidence 0.13
First Reported 1999
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 3.72
Pain Relevance 0.72

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell proliferation (RETNLB) extracellular region (RETNLB) molecular_function (RETNLB)
cellular_component (RETNLB)
Anatomy Link Frequency
plaque 1
saliva 1
blood cells 1
RETNLB (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 95 100.00 Very High Very High Very High
cytokine 11 99.96 Very High Very High Very High
Pain 1 85.04 High High
rheumatoid arthritis 1 76.72 Quite High
Inflammatory mediators 4 57.52 Quite High
Angina 1 15.44 Low Low
Inflammatory response 5 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
cINOD 1 5.00 Very Low Very Low Very Low
chemokine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 105 100.00 Very High Very High Very High
Disease 40 99.96 Very High Very High Very High
Atherosclerosis 13 99.92 Very High Very High Very High
Dengue Hemorrhagic Fever 205 96.92 Very High Very High Very High
Rupture 4 96.92 Very High Very High Very High
Mucositis 6 95.64 Very High Very High Very High
Sepsis 6 94.48 High High
Bacteremia 1 94.08 High High
Stress 10 91.96 High High
Cancer 1 87.64 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The condition appears to represent a sequential interaction of the oral mucosal cells and tissues, pro-inflammatory cytokines, and local environmental factors in the mouth such as microorganisms and saliva.
pro-inflammatory Binding (interaction) of in saliva associated with inflammation and cytokine
1) Confidence 0.13 Published 1999 Journal Leukemia Section Abstract Doc Link 10360368 Disease Relevance 0.97 Pain Relevance 0.22
The fact that atherosclerosis is recognized as an inflammatory disease, along with the correlation observed between plaque temperature and rupture likelihood, is awakening interest in the determination of the arterial wall temperature (AWT) and the characterization of plaque vulnerability through local temperature [21].
inflammatory Binding (recognized) of in plaque associated with inflammation, rupture, atherosclerosis and disease
2) Confidence 0.01 Published 2007 Journal Biomed Eng Online Section Body Doc Link PMC1838421 Disease Relevance 1.30 Pain Relevance 0.12
Activation of a pro-inflammatory defence gene pattern in DSS patients' blood cells (Table 4) has relevance to the pathophysiology of systemic vascular dysfunction, since most microbicidal peptides and enzymes have recognized pro-inflammatory and pathogenic effects towards vascular endothelial tissues [50].
pro-inflammatory Binding (recognized) of in blood cells associated with inflammation and dengue hemorrhagic fever
3) Confidence 0.01 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2907396 Disease Relevance 1.45 Pain Relevance 0.37

General Comments

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